PLACE IN RETURN BOX to remove this checkout from your record. TO AVOID FINES ratum on or before date due. DATE DUE DATE DUE DATE DUE I MSU Is An Affirmative Action/Equal Opportunity Institution A VISUAL HIEIDHIC VERSUS DIRECT IISTRUCTIOH APPROACH TO FACILITATE RECALL II CLOSED HEAD IIJURY PATIEITS BY Angela R. lbssenberg A DISSERTATION Submitted to Hichigan State University in partial fulfillnent of the require-ants for the degree of DOCTOR OF PHILOSOPHY Department of Audiology and Speech Sciences 1988 ABSTRACT A VISUAL HNEMONIC VERSUS DIRECT INSTRUCTION APPROACH TO FACILITATE RECALL IN CLOSED HEAD INJURY PATIENTS BY Angela R. Massenberg Two experiments were conducted to determine the effects of mnemonic based instruction versus direct instruction on the recall of personal names by individuals with a Closed Head Injury. Experiment I utilized an alternating treatment, single-subject multiple baseline across subjects design in which four adults served as subjects. Each subject was taught a total of 20 personal names, ten with a mnemonic based instruction and ten with a direct instruction approach, to determine which type of instruction would yield better recall of the stimulus names. Each treatment session consisted of the following procedure: Initially, each subject was shown photographs of individual people whose name was to be recalled. Those names receiving the mnemonic based instruction were paired with a second photograph which depicted the individual holding an object that was representative of the person’s name (i.e., a mnemonic). Those photographs receiving the direct instruction approach depicted the individual without the mnemonic. The results revealed that two out of the four subjects demonstrated improved name recall with mnemonic instruction, while the remaining two subjects demonstrated no marked improvement in name recall under either instructional mode. Experiment II employed an alternating treatment single- subject multiple baseline across behaviors design with two subjects. The treatment procedure was the same as in Experiment I, except that in Experiment II each subject served as his own control. Results from Experiment II revealed that both subjects demonstrated a trend for better recall of personal names receiving the mnemonic instruction as compared to those receiving the direct instruction approach. To my beloved mother, Hallie Hassenberg ii ACKNOWLEDGEMENTS Many fine people have assisted me and provided support throughout my doctoral program. I would like to thank the following people: Dr. Michael W. Casby, my academic advisor and major professor, for his abiding support, interest, encouragement, and knowledge that he so willingly shared. He has taught me more than I could ever express, and for that I am so grateful. Drs. Richard Johnson, Peter LaPine, and John Tonkovich, for their willingness to serve on my committee and the guidance provided during the preparation of the study. Ms. Charlene McNeal and Ms. LaVerne Moore, Administrators, of the Irvine Group Home, for the expeditious manner in which they secured three of the subjects for participation in my study. Dr. Jeffrey Ditty and Ms. Terri King, Administrators, of J.A. Ditty and Associates, Inc., for their willingness to allow me to utilize two of their clients for participation in my study and the expeditious manner in which the subjects' participation was secured. iii Dr. Timothy Broe, Administrator, of Broe Rehabilitation Services, for his willingness to secure one of his client’s for participation in my research. Ms. Juanita McKinnie, who served as a research assistant, and provided constant encouragement. All the people who agreed to participate in the study. Finally, I want to thank my family who has been my anchor throughout my educational pursuits. I especially want to thank my father, Mr. Millard F. Massenberg, Jr., who has constantly been there for me, for all the love he has always given, and for his pride in me that he has always openly expressed. He taught me the meaning of perseverance, and having his continuous support and encouragement has meant so much to me. I also want to thank my sister, Ms. Dianne Frazier, and my grandmother, Ms. Rebecca Massenberg (bereaved), for their love, continuous support, and encouragement. iv TABLE OF CONTENTS LIST OF TABLES .................................. LIST OF FIGURES ................................. LIST OF APPENDICES .............................. INTRODUCTION AND REVIEW OF THE LITERATURE... METHOD: Experiment I ........................ Subjects ............................... Experimental Design and Procedures ..... Treatment .............................. Reliability ............................ RESULTS: Experiment I ....................... Subject 1 .............................. Subject 2 .............................. Subject 3 .............................. Subject 4 .............................. METHOD: Experiment II ....................... Subjects ............................... Experimental Design and Procedures ..... Treatment .............................. Reliability ............................ Page vii viii ix 24 24 32 38 42 43 46 47 49 50 51 56 57 61 Page RESULTS: Experiment II ....................... 62 Subject 5 ............................... 62 Subject 6 ............................... 66 DISCUSSION .................................... 71 REFERENCES .................................... 78 APPENDICES .................................... 88 vi LIST OF TABLES Table Page 1 Rancho Los Amigos Scale of Cognitive Levels and Expected Behavior ........... 29 2 Functional Communication Measure: Cognitive Communication ................ 31 3 Description of Subjects for Experiment I ........ . ................. 32 4 Pretest Recall Results for CHI Subjects ............................... 36 5 Recall Results for Non-brain Damaged Subjects ............................... 37 6 Description of Subjects for Experiment II .......................... 55 7 Pretest Recall Results for Subject 5 and Subject 6 .......................... 58 vii LIST Figure 1 Baseline and treatment for Subjects 1 and 2 Baseline and treatment for Subjects 3 and 3 Baseline and treatment for Subject 5 ..... 4 Baseline and treatment for Subject 6...... ............... OF FIGURES data data viii Page 44 45 63 67 Appendix A Stimulus B C D Subtest E Subtest F Subtest G Subtest H Subtest I Subtest J LIST OF APPENDICES Names ............................. Boston Diagnostic Aphasia Examination Results for Subjects 1 - 4 ............... Boston Diagnostic Aphasia Examination Results for Subjects 5 and 6 ............. Summary Summary Summary Summary Summary Summary Profile Profile Profile Profile Profile Profile for for for for for for Subject Subject Subject Subject Subject Subject 1 2 5 6 000000 Informed Consent Form ...................... ix Page 88 89 93 97 98 99 100 101 102 103 Introduction Every year hundreds of thousands of people are involved in accidents. Often times, the result of these accidents, is a Closed Head Injury (CH1). CH1 can be defined as a non- penetrating traumatic injury to the head that results in cerebral dysfunction (Adamovich, et al, 1985). According to the National Head Injury Foundation, Inc.,the estimated prevalence of head injuries in the U.S. is 1,000,000- 1,800,000 (Williams, 1986), with 350,000 resulting from automobile accidents. It has been further estimated that 140,000 Americans die annually from a head injury, and that 20,000 remain comatose (Williams, 1986). Additionally, head injuries are responsible for up to 60% of auto-related deaths (Pederson, 1986). Due to medical advances (e.g., improved emergency room procedures, medical technology) many more of these victims, especially with severe head injuries, are surviving (Dikmen et a1, 1983; Kirn, 1987). Approximately 50,000 - 70,000 CHI victums are left with permanent physical and intellectual disabilities however (Williams, 1986). These residual deficits include physical handicaps, memory impairments, attentional problems, impaired cognition, visual and auditory difficulties, as well as difficulties with communication (Levin, 1986; Adamovich et a1, 1985). Neuroanatomical and Neuropsychological Changes The individual sustaining a closed head injury suffers from many neuroanatomical and neuropsychological changes; this is particularly true with severe cases. A severe blow to the head results in cortical neuroanatomical alterations caused by the brain’s accelerating, rotating, compressing, and expanding within the skull (Hagen, 1981; Hooper, 1969; North, 1984; Russell, 1971). When the brain accelerates, and ultimately decelerates, as a result of a blow to the head, there is only a limited extent in which it may move within the skull and dural envelope. When this movement is suddenly ceased (due to the frontal and temporal poles impacting against the walls of the anterior and middle cranial fossae), damage is done to the tips (poles) of the temporal and frontal lobes, as well as to the orbital surface of the frontal lobe (Auerbach, 1983, 1986; Miller, 1983; North, 1984). The rotating movement of the brain results in widely shattered shearing of axons within the myelin sheaths leading itself to diffuse brain damage. The jarring movement of the brain itself also results in cortical contusions and/or lacerations. Cortical Damage As a result of the cortical damage resulting from a CHI there are various physical manifestations (e.g., cognitive, behavioral, and emotional changes; memory problems, agnosia, apraxia, aphasia and related speech/language problems; visual field disturbances; visual spatial disturbances; hemiplegia, hemipareses; cortical blindness, seizures, perseveration) (Baggerly, 1986). Some manifestations of a CHI may be the result of emotional and behavioral alterations that occur with damage to the medial cortical regions that are deemed part of the limbic system. For example, arousal and attentional deficits may be the result of diffuse white matter injury which is associated with altered consciousness. Frontal Lobe. The frontal lobe area of the brain contains the precentral cortex which controls all motor functions. A localized lesion in the posterior inferior region results in difficulty in programming and direcing the speech musculature in the intergrated coordinated sequences necessary for speech (i.e., apraxia of speech) (Brookshire, 1978). Lesions in the same area may result in a Broca's aphasia. Characteristics of Broca's aphasia include nonfluent speech, consisting of a few words and short sentences (Kertesz, 1979; Damasio, 1981). Speech production is laborious and telegraphic. However, auditory comprehension is relatively intact. There are a host of other behaviors that are particularly observed with those individuals who have sustained frontal lobe injuries secondary to a CHI. Among these are personality changes, either being more subdued or aggressive than was the case premorbidly (Baggerly, 1986; Habermann, 1982; Oddy & Humphrey, 1980). Also, signs of euphoria or apathy, lack of insight, lack of social graces or disinhibition and inability to self-monitor, emotional responses that are inappropriate for a particular situation are observed. It has also been observed that some CHI individuals demonstrate a lack of initiative and goal- directed behavior. These individuals in general, demonstrate poor problemrso1ving/reasoning skills, with responses being concrete and literal. They also often exhibit difficulty with cognitive flexibility, that is, "shifting from one concept to another" (Baggerly, 1986), as well as perseveration. Parietal Lobe. Resultant parietal lobe damage from a CHI involves many of the cortical sensory and sensory intergration systems which are altered as a result of a CHI. Specifically, deficits in the ability to identify familiar objects based on tactile information, that is, asterognosis, may be observed. In the most general sense, damage to the left hemisphere many result in aphasic and problems such as alexia (i.e., difficulty with reading information) and agraphia (i.e., relative to the impaired spelling). The portion of the angular gyrus that is within the parietal region has sustained damage to note the alexia and agraphia (Baggerly, 1986; Smith, 1979). Skilled movement deficits and/or reduced analytical and logical reasoning abilities may also be impaired as a result of parietal lobe damage. Constrastly, right hemisphere parietal lobe damage will typically result in visual-perceptual and spatial impairments, agnosia and loss of prosody (Baggerly, 1986). Temporal Lobe. Damage to the dominant temporal lobe in general, results in reduced language abilities. The second most common language disorder after a CHI is Wernicke’s aphasia (Levin, 1981), which is located in the posterior aspect ofthe temporal region. The characteristics observed with this language disorder consist of fluent, well articulated paraphasic speech, poor comprehension for oral and written language, and impaired repetition (Brookshire, 1978; Kertesz, 1979). Furthermore, a CHI with damage to the temporal area produces behavior manifestations such as anxiety, calmness, rage, aggression, and depression. These behaviors are triggered by internal mechanisms and are not asssociated with circumstances in the external environment. The structures involved include the amygdala which is responsible to a large part for these various emotional responses (e.g., rage, aggression) (Heimer, 1983). When considering the neuropsychological problems which occur as a result of a CHI, the temporal lobe is also an important region for memory/new learning (Arnold, 1982; Khan, 1986). The hippocampi, a structure connected with recent memory is located medially to the temporal lobe, is frequently damaged (Muramoto et al., 1979; Pang, 1985). The hippocampus although important for recent memory (i.e., facts that are presented or events that occur after the onset of damage-anterograde, Nissen, 1986), has connections/ circuits with other types of memory, as well. ggmory Functioning in Non-Brain Injured Individuals Multiple Stores Theory When considering normal memory functioning the multiple store theories would be appropriate. These theories postulate that there are two or more functionally different types of memory systems (Howard, 1983; Reynolds & Flagg, 1977). The two different types of memory storage, consists of one that is transitory, and the other, permanent. The two major types of memory are short term memory, also known as working memory, and long term memory, or reference memory (Howard, 1983; Reynolds & Flagg, 1977; Somjen, 1983). The function of STM is thought to be where information is ititially held, however, remaining there anywhere from a few seconds to 1-2 minutes. This memory has a small capacity, being able to accommodate a maximum of seven items, plus or minus two, at any given time (SomUen, 1983). To retain information at this level it must be actively rehearsed. Long term memory possesses an extremely large capacity of memory traces. Somjen (1983) decribes memory traces as the elementary form of information storage in the brain. These memory traces are more difficult to acquire than those of STM, however once obtained, are sustained for a indefinite amount of time, perhaps the lifespan of the individual. When information in LTM can not be readily retrieved, it is not a result of the memory trace fading, but rather access to it may be hindered. LTM acts as an enormous warehouse that stores all that one knows, ranging from vocabulary to the content of various life experiences. When the information from the sensory store is matched, the meaning or ”name" associated with the LTM represents a transfer to STM; however information may go directly to LTM. Howard (1983) discusses a memory system that consists of three stores, in which STM as described aboved is broken into two parts, sensory registers and working (i.e., STM) memory, and LTM. At any one time many stimuli impinge on one’s sensory organs, with these representations residing briefly in the sensory registers. It is assumed that each of these representations automatically contacts a place in long term memory where information about the stimulus is housed. It is thought that part of the LTM representation of an item is automatically contacted whenever that item is placed in the sensory register, but we do not assume that a permanent record of that occurrence is stored in LTM. New information is only stored permanently in LTM, if further attention is given to the item. Neurophysiological Bases of Memory There are many theories that attempt to explain the storage of memory traces. It has been suggested that there are neurophysiological differences between STM and LTM. With STM, a form of electrical activity among neurons occur (Reynolds & Flagg, 1977). However, LTM due to its longevity, seems to involve either structural changes among neurons (e.g., new interconnections are developing) or the formation of new chemical molecules, or both. Somjen (1983) discusses various theories of memory trace formation, with the two most recent ones being discussed. Theories of eeuipotentiality‘and of mass action Somjen (1983) addresses Lashley's search for the engram (i.e., memory trace) in experiments in which various parts of the cerebral cortex of animals were surgically removed. The animals were tested for three behaviors: 1) retention of habits learned before the animal’s operation; 2) their ability to re-learn what they appeared to have forgotten; and 3) their ability to learn new tasks. It was concluded that memory function is disturbed in proportion to the amount of cortx destroyed, irrespective of which part of the cortex had been removed. As far as learning was concerned, it was thought that all parts of the cortex were equipotent, with the exception of the visual cortex. Instead of localized storage of memory traces, Lashley proposed a new concept. He agreed that recall entails reactivating a previous pattern of neuronal excitation. However, he argued that the pattern representing any one memory could be evoked not in just one specific set of neurons. Lashley believed that many sets in many places could be evoked, and perhaps anywhere in the areas of the brain that have to do with memory function. Theories of chemical coding It has been proposed that the experiences acquired by an individual could be stored in the brain in ways similar to that by which the experiences of the species are laid down in genetic information in cell nuclei, out of which grew the theory of encoding memories in the structure of macromolecules (Somjen, 1983). The nucleic acids or proteins were the only molecules that could serve as chemical encoders due to their size and structure. Somjen (1983) notes that if this theory were true many secondary problems would arise. For instance, how would the sensory information which is originally encoded in neuronal signals be translated into the chemical code and how would memory traces stored in chemical form be re-translated into neuronal signals when they were recalled? Moreover, would all the information one has learned be stored in all the neurons of the brain, or is learning domain of specialized 10 cell groups? Are certain kinds of memories stored in specific types of neurons? Many experiments have been conducted to attempt to answer these questions. However, this issue has remained unresolved. In summary, for what is known today, it is not possible to determine whether all forms of learning depend on variants of a single kind of elementary biophysical or biochemical change, or whether there exists several basically different mechanisms of memory trace formation. Memory and CH1 One major neuropsychological impairment demonstrated by CHI individuals, which has been well documented is that of memory deficits (Adamovich et al., 1985; Arnett & Corrigan, 1986; Auerbach, 1983, 1986; Baggerly, 1986; Brooks, 1972, 1974, 1976, 1983; Brooks et al., 1980; Evans, 1981; Fodor, 1972; Glasgow et al., 1977; Gloag, 1985; Hagen, 1981; Mandleberg & Brooks, 1975); McMillan & Glucksman, 1987; Pederson, 1986; Richardson, 1979; 1984; Schacter & Crovitz, 1977; Thomas & Trexler, 1982; Violon & DeMol, 1987). Closed head injury patients demonstrate difficulty with remembering new information. In fact, the most apparent memory deficit in the severely brain-injured individual is the extreme difficulty encountered in retaining new information (Arnett & Corrigan, 1986; Nissen, 1986). Pang (1985) and Somjen (1983) both noted that although new information is correctly perceived, it can not be stored beyond immediate use. This deficit was interpreted as an inability to transfer information from short term to long term memory, that is, to consolidate memory traces. It was also reported that the most prominent memory deficits are the loss of the ability to recall numbers, events, and names several minutes after learning them (Somjen, 1983), although immediate retrieval is generally possible (Pang, 1985). 11 12 Violon & DeMol (1987) investigated the memory functioning of CHI patients, as assessed by the Rey's 15 words test. Although no description of this test was provided to assist in determining what the task entailed, these researchers found that the memory functioning was altered in 64% of CHI patients during the first weeks following the trauma. At six months post-injury, memory remained impaired in 48% of the cases. When considering those patients who sustained a severe CHI, Violon and DeMol (1987) reported that none were free of memory disturbance during the first month. Their data also showed that five months after the injury, only 6% patients had normal memory, with only 12% showing normal memory within the first 2 years, and 21% after 2 years. Also, Pederson (1986) found that of 252 CHI subjects, 82% had memory problems. Such staggering data regarding memory disturbances clearly emphasizes the prevalence, persistence and magnitude of this deficit. While Violin & DeMol (1987) and Pederson (1986) reported that individuals with severe CHI demonstrated memory problems, Richardson (1979, 1982, 1984) reported that CHI patients with minor head injuries also present memory problems. In 1979, he investigated 48 patients diagnosed as having a minor CHI (i.e. less than 8 hours from trauma to return of continuous, day-to-day, memory) to determine if 13 there was a difference in their recall of concrete and abstract words. He investigated the use of these two types of words because he hypothesized that CHI patients were impaired in their ability to recall common nouns, but that this was a result of a defect in imaginal encoding. Therefore, the impairment of memory function should be with concrete material but not with abstract material. In testing his hypothesis, Richardson employed a total of 100 nouns. Fifty of these nouns had imagery and concreteness ratings of 6.0 or above, based on Paivio and associates’ research (1968), (e.g., alligator, baby); while the remaining 50 nouns had such ratings of less than 6.0 (e.g. answer, anecdote). The subjects were presented with 10 lists, each with 10 words. The words were presented at a rate of one every 3 seconds, with 30 seconds given for the immediate oral recall of each list. Following the recall of the 10th list, each patient was instructed to recall all 100 words within a 3 minute time limit. It was found that the CHI the patients showed a significant reduction in the recall of concrete words when their responses were compared the control group of subjects. The normal control group did not perform better than the CHI subjects with the abstract words, however. The control subjects showed the normal advantage in the recall of concrete material over that of abstract material, but the 14 CHI subjects did not. Due to both groups of subjects performing the same with the abstract words, Richardson argued that his present investigation failed to find any suggestion of an impairment in the recall of abstract words for the CHI subjects. Furthermore, the CHI group showed no significant difference between the two sorts of material. It was concluded that the major finding in this study was thus of a specific impairment in the recall of concrete words with CHI patients. Furthering his 1979 investigation, Richardson (1984) examined the types of errors made by CHI patients when attempting to recall lists of concrete nouns. He wanted to determine if the subjects made intrusive errors of words from previous lists; or if they made phonemically related errors (e.g., tar for car) or semantically related errors (e.g., chair for table). Richardson (1984) found no evidence to support that a CHI affects the occurrence of either phonemic or semantic related confusion errors. He did report however, that the CHI subjects did make intrusive errors. He further noted that the basis of the intrusion errors of the CHI subjects, unlike those of the control group, were meg related to the image evoking feature (i.e., the level of concreteness) of the such stimulus items. When recalling words from the concrete lists, the control group made 58 concrete intrusion errors (i.e., those 15 58 errors were from other concrete lists than the list that was being assessed at that time), but only 20 of their intrusion errors were from previous abstract lists. In other words, the control subjects were recalling more concrete words, although they were not recalling the correct concrete words for the appropriate concrete list. When the CHI subjects recalled the same concrete lists, they made 37 concrete errors, and 46 abstract intrusion errors. That is, unlike the the normal group who relied on the image-evoking quality to recall the concrete words (as evident by their 58 errors), the CHI subjects were not attending to the image- evoking quality that would assist in recalling these concrete words. The control group demonstrated a clear relationship between the concreteness of the list from which an intrusion error apparently originated and the concreteness of the list from which the subject was attempting to recall. Findings for the control group demonstrated that the image-evoking quality for the noun was the highly salient feature in deciding whether or not an item was retrieved. However, the CHI subjects did not demonstrate such an effect. The concreteness of their intrusion errors was essentially unrelated to the concreteness of the list from which they were attempting to recall. Richardson suggests that the CHI patients do not attend to the concreteness or image—evoking aspect of the current list in evaluating which items to It? (f 16 retrieve. It was therefore suggested that there may be a disruption in the CHI patients’ employment of mental imagery as a form of elaborate encoding in long term memory. Richardson argued that this might be attributed to a failure to attend to the image-evoking quality of the stimuli. An implication of the Richardson research is that the memory deficit resulting from a CHI might be alleviated if the image-evoking quality of the stimulus material is explicitly indicated or enhanced for CH1 patients with memory problems. Hence, as suggested by Richardson (1984), although CHI patients may not spontaneuously use imagery they may be taught to use such a strategy, and thereby ameliorate their memory problems. Utilieing Mnempnic Strategies with CHIgpatieQEe As stated earlier, memory deficits is one of the major neuropsychological impairments present following a CHI. What has been less researched, however, is how to treat the memory deficits that are present in such a way to improve the functioning of these individuals. Strategies utilized to facilitate memory range from verbal rehearsal to visual mnemonics. Glasgow et al. (1977) reported a verbal rehearsal strategy called PQRST. The acronym represents the following steps: Preview (i.e., briefly skim the material to learn for its general content); Question (i.e., ask key questions relative to the content); Read (i.e., read 17 actively with the goal of answering the questions); §£fl£§ (i.e., repeat or rehearse the information that has been read); and Tee; (i.e., test oneself by answering the questions that have been developed). By virtue of its format, this approach utilizes a rehearsal strategy, requiring the individual to state aloud what was recalled about the stimulus material. Examples of the visual mnemonic approach could be the keyword and pegword methods. Scruggs et al. (1987) studied the use of mnemonic facilitation of LD students’ memory for expository prose. Two experiements were conducted. With one, the students were seen individually and were read a passage describing three types of dichotomous information about eight North American minerals. Such attributes as hardness (soft vs. hard), color (pale vs. dark), and use (home vs. industrial) were included. The eight stimulus- mineral words (and the keywords in the mnemonic condition) were then presented from a list. The mnemonic pictures exerted a strong facilitative effect on LD students’ learning of textually embedded science facts. During Experiment 2, the students were explicitly instructed to remember specific attributes rather than dichotomous atttribute classifications. In the mnemonic condition, a list of 10 rhyming "pegword" (e.g. eee_is 233, Eye is eeee, etc) was used to represent the Hardness levels 1 through 10. THe instructions indicated that the children 18 would be using ”word clues" (keywords) and "number clues" (pegwords) to recall speicific hardness-level, color, and use attributes of eight minerals. Using the mineral crocoite, the picture included an orange (color) crocodile (keyword for crocoite) wearing shoes (pegword for Hardness Level 2) in a display case (used for displays). Also, the materials in the control condition paralleled the mnemonic materials except that the keywords and pegwords were not included, and nonmnemonic illustrations were used for representing each mineral’s hardness level, color, and use. Results revealed that for all four attribute-memory tests given, the mnemonic subjects outperformed control subjects. In particular, the visual mnemonic strategy has been applied in a number of different ways with closed head injury patients. One approach is based on a paired association strategy in which a patient is required to generate a visual image of, for example, a the person’s name. Glasgow, et al. (1977) discussed a case study in which such a procedure was used with one head injured patient. The patient was taught to remember names of people from a list of names that he wanted to remember. The patient wrote the names on a 3x5 card and three times daily, he read each name on the list. He also was instructed to visualize the people as he read the names to be remembered. It was reported that the patient did note improvement. However, 88: of vex efi pai The in: ma: (V: ha' am re] the the Fe: Ha: 11a: in. Ed: f9,- 0f HO! 19 self-reports were the only measure used to assess the effect of treatment. Wilson and Moffat (1984) cite studies in which two versions of visual imagery were reported to be used effectively to facilitate memory for names with head injury patients, as well as with other memory impaired individuals. The first strategy involved having the patient form a mental image associated with someone’s name. For example, if the name Angela Webster was to be learned, the patient would construct a mental image involving an angel, web, and star (Wilson & Moffat, 1984). This system has been reported to have been successfully applied to learning names of staff and patients by amnestic individuals attending rehabilitation centers (Wilson & Moffat, 1984). However, the subject population of this report was not specific, and the methodology portion provided insufficient information. Another approach discussed by Wilson and Moffat requires subjects to image a feature of the person whose name is to be remembered to assist in the recall of the name. It is coined face-name association. The method involves linking a distinctive feature of a person with the name which is to be mentally imagined (Glasgow et al., 1977; Wilson & Moffat, 1984). For instance, if the distinctive feature of Angela Webster was her hair, the image might be of angel and a star caught in a web, made out of hair. However, it appeared that certain brain damaged people were 11 "U "U 0) *ri 20 unable to cope with all the stages of this strategy (Wilson & Moffat, 1984). Again, however, subject description was inadequate, making it impossible to discern which brain damaged people experienced the most difficulty. With such strategies it must be noted that there are elements of rehearsal and paired associate learning, as well as visual mnemonics. These techniques differ on the reliance of external and self-generated cues. Wilson (1987) using a multiple baseline across behaviors design, investigated the use of visual imagery versus rehearsal with a CHI subject, who was being taught 16 personal names of staff personnel whom he was familiar. Photographs were taken of the 16 people and paired with line drawn pictures of mnemonics which represented each name (e.g., the name Mike may be depicted as a man holding a microphone). All 16 stimulus names were held in baseline, and every day or two two new names were introduced, one for the visual imagery condition, and one for the rehearsal condition. Recall was assessed after 2 minutes, 30 minutes, and 24 hours. During recall each of the 16 photographs were placed before the subject, and he was asked to identify the person’s name. Any mistakes were corrected but scored as errors. The results revealed that the visual imagery condition was significantly better than rehearsal based on a Wilcoxon analysis (p < .01). Overall, the subject’s best recall 21 performance was 6 out of 8, and by the final session (i.e., session 22), there was a slight decrease to a recall level of 5 out of 8. In the rehearsal condition, the subject recalled only 2 out of the 8 names. It should be noted that the investigator did not indicate which recall results (i.e., 2 minutes, 30 minutes, or 24 hours) were being recorded. Furthermore, the multiple baseline across behaviors design was unorthodox, in that new names were trained every 1-2 days regardless of whether or not improvement in the recall of previous names was noted. Massenberg and Casby (1988) reported two single - subject multiple baseline studies in which a visual mnemonic strategy was employed to assist CHI patients’ ability to remember personal names. The strategy involved presenting a visual mnemonic of a name which was to be remembered. The training package for the first subject consisted of a total of 13 photographs including 5 of the professional staff, and 8 of contemporary celebrities, while for the second subject, 15 photographs, 8 and 7, respectively. For each stimulus photograph a black line figure was drawn which represented a visual mnemonic for the name of the person. For example, the name "Claudia" was drawn as a elem and a gee (bag), and "Therese", as two "trees". This visual mnemonic was then paired with the respective photograph and presented to the subject. During the first three baseline evaluations subject 1 DQ‘ am 1'9! SE tr (1 ar ti t1 t1 Té 22 never immediately named more than 2 of the 7 set one names and no more than 2 of the 6 set two names. He initially received treatment for the recall of the set one names, while the second set of names were continued in baseline. The subject 2 never named more than 1 out of 8 for the set 2 names, and 1 out of 7 for the set 1 names. The training package was introduced in a counterbalanced order (i.e., for subject 2, set 2 first, followed by set 1). Results showed that both of the subjects made immediate and steady progress when the treatment was applied to the the first set of names trained, i.e., staff or celebrity names. Both subjects made marked gains in each of the first three treatment sessions with continued improvement thereafter. While improvement was being made with the recall of the first set of names trained, the subjects continued to perform poorly on baseline measurements of recall of the second set of names. It was not until session 9, when the treatment was applied to the second group of names, that the subjects showed marked improvement in their recall. After a five week training period results demonstrated that both of the subjects had increased their correct recall of personal names to a level of 92%, and 93%, respectively for the two subjects. Memory deficits resulting from a CHI are quite prevalent and persistent. Memory functioning is affected to some degree for the majority of the CHI patients. C01 PO] of ex! de 1a 81 be at tc 11 23 Considering the magnitude of this problem in the CHI population, it is unfortunate that there is only a paucity of clinical research available which addresses treatment of these memory deficits (Levin et al., 1982). With few exceptions the treatment studies reported in the literature demonstrate poor methodology, a lack of empirical support, a lack of, or poor controls, and no replication data for single case studies. The memory strategies discussed generally included elements of rehearsal and paired associate learning, as well as visual mnemonics, with no attention to the individual additive contribution of each of these. Furthermore, little controlled clinical research has followed-up on Richardson’s suggestion that CHI patients may be taught to attend to the image-evoking aspect of stimuli and thus improve there memory. The purpose of this study is to take into account the aforementioned observations. This investigator wanted to determine the effect of visual mnemonic instruction, on the memory of the CHI individuals. Experiment I Method An alternating treatment, single subject multiple baseline across subjects design was utilized to investigate the efficacy of a direct instruction based intervention approach versus a mnemonic based instruction approach when teaching personal names to patients with CHI. Subjects Four adults, who had sustained a CHI, served as subjects. The ages ranged from 23 - 41 years. The subjects had no history of previous head injuries or other neurologically based disorders, as well as no history of alcoholism or substance abuse as determined by case history interviews. The subjects were functioning minimally at cognitive level VI, Ranchos Los Amigos Scale (Hagen, 1981), and no higher than a cognitive level VII, as confirmed by the investigator. As can be seen in Table 1, at this level the patient is able to demonstrate carryover of learned tasks, and is able to exhibit goal directed behavior, although still dependent on external input for direction (Adamovich et al., 1985; Baggerly,1986). Based on the Functional Communication Measure - Cognitive Communication (Larkins, 1987), the subjects were at least functioning at a Level 4 but no higher than a Level 5 (see Table 2 for entire 24 25 scale). The subjects’ range for time since CHI was from 1 year to 12;7 years. Subject 1 was a 41 year old female who was employed for 20 years at a major insurance company. She was involved in an automoblie accident, sustaining a CHI one year prior to her participating in this study. At the time of the research, she was currently taking the medication, Tranxene, 3.75 mg. daily, which is a tranquilizer. Subject 2 was a 23 year old male who also sustained a CHI secondary to an automobile accident. His accident occurred 6;3 years prior to participating in this study, while he was a senior in high school. During the first eleven training sessions of the research the patient was taking 800 mg. Tegratol, 300 mg. Dilantin, and 150 mg. Buspar, all to control seizures. During the twelfth through sixteenth session his medication regime increased to 1000 mg. Tegratol, 400 mg. Dilantin, and 150 mg. Buspar. Subject 3 was a 29 year old male. At the time of his accident, 1;10 years prior to his involvement in this study, he was employed at a major automotive factory. The subject’s CHI resulted from his being struck by an automobile. At the time of the research he was currently on no mediation. Subject 4, also a 29 year old male, sustained a CHI at the age of 17 years. He was a senior in high school, when 26 involved in an automobile accident. At the time of the study he was taking 25 mg. of Mellaril daily, to reduce anxiety (i.e., a tranquilizer). The subjects were given the Boston Diagnostic Aphasia Exammnation (BDAE) (Goodglass & Kaplan, 1983) to assess language abilities. This test examines the areas of auditory and reading comprehension, oral expression including repetition, naming, oral reading, and automatic speech abilities, paraphasia and fluency, writing, and recitation, singing, and rhythm. In evaluating the subjects performance results the BQAE cut-off scores for normal performance and the percentile ratings based on the aphasic population were used. Subjects 1 and 3 were found not to fall below the cut-off for any of the subtests of the BDAE. Subject 2 fell below the cut-off score in the following areas (see Table 3 for the total number of subtests that fell below the cut-off scores): Auditory Comprehension - Word Discrimination with a raw score of 64 (cutoff score = 67), ranking at the 70th percentile, Animal Naming with a raw score of 11 (the cutoff score = 12), ranking at the 9lst percentile, and Comprehension of Oral Spelling, with a raw score of 5 (the cut-off score = 6), ranking at the 75th percentile. Subject 4 fell below the cut-off score for normal performance for repetition of Low Probability Sentences with a raw score of 5 (the cut-off score = 6), ranking at the 27 85th percentile, and for Reading Sentences and Paragraphs with a raw score of 6 (the cutoff score = 7), ranking at the 70th percentile. (See Table 3 for the total number of subtests that fell below the cut-off scores). Overall, none of the subjects demonstrated any specific aphasia syndromes. (See Appendix C for the raw scores and percentile ranks for all major areas for Subjects 1 through 4, and Appendices D, E, F, and G, for Subjects’ 1 through 4 individual Subtest Summary Profile, respectively). The Woodcock-Johnson Psycho-Educational Battery (Woodcock & Johnson, 1977), Memory Cluster subtest, was given to assess the subjects’short term memory abilities. This subtest consists of two parts. One requires that the individual repeat sentences of varying lengths. The second portion requires one to repeat digits in a reversed order. Subjects 1 through 4's performance ranged from the 15th percentile to the 24th percentile (see Table 3). The Airplane List (Crovitz, 1979) was also used to assess the subjects’ memory. It consists of a story passage where throughout, the individual is told to remember a total of 10 words. The Airplane List is based on mnemonic encoding principles. Recall of the ten words was assessed immediately, and with a 30 minute delay. As can be seen from Table 4, Subjects 1, 2, and 3 were each able to recall four items immediately, whereas Subject 4 recalled two words immediately after presentation of the story. After 28 the thirty minute delay, recall performance decreased to 2, 0, 3, and 1, for Subjects 1 through 4, respectively. Crovitz (1979) found that when the Airplane List was given to non-brain damaged individuals they were able to recall all ten words in order forward and in reversed order. Radiological information was available for three of the four subjects. Electro-encephalography information was available for Subject 1 revealing left temporal lobe damage. CT information for Subject 2 revealed no specific site(s) of lesion, but rather overall cerebral atrophic changes. Subject 3’s most recent CT scan (within a year of this experiment) was normal. No such radiological information was available for Subject 4. All the subjects were screened for prosopagnosia (i.e., the inability to recognize familiar faces). This was done by having the subjects match color facial photographs of various individuals in two different poses (i.e. full-face to profile). All subjects' passed with 100% accuracy. In addition, each subject passed a hearing screening evaluation at 25 dB HTL, at 250, 500, 1000, 2000, 4000, and 8000 Hz. Inquiries were made regarding the subjects’ wearing of eyeglasses, and if so, they were worn during treatment. 29 Table 1 Rancho Los Amigos Scale of Cognitive Levels and Expected Behavior Associated with Closed Head Injury Level I No Response Unresponsive to all stimuli Level II Generalized Inconsistent, nonpurposeful, Response nonspecific reaction to stimuli. Responds to pain, but response may be delayed. Level III Localized Inconsistent reaction di— Response rectly related to type of stimulus presented. Re~ sponds to some commands. May respond to discomfort. Level IV Confused, Disoriented and unaware of Agitated present events with Response frequent bizarre and in- appropriate behavior. Attention span is short and ability to process information is impaired. Level V Confused, Nonpurposeful random or Inappropriate fragmented responses when Nonagitated task complexity exceeds Response abilities. Patient appears alert and responds to simple commands. Performs previously learned tasks but is unable to learn new ones. Level VI Confused, Behavior is goal-directed. Appropriate Responses are appropriate Response to the situation with in- correct responses due to memory difficulties. More awareness of not knowing the correct answer. Completes previously learned tasks with supervision. 30 Level VII Automatic, Appropriate Response Level VIII Purposeful, Appropriate Response Correct routine responses which are robotlike. More awareness of self and others. Appears oriented to setting, but insight, judgement, and problem- solving are poor. Correct responding, carry- over of new learning. No required supervision, poor tolerance for stress, and some abstract reasoning difficulties; more awareness of deficits Adamovich et al. 1985; Baggerly, 1986 (Scale originally developed by Hagen, C., and Malkmus, D., 1979) 31 Table 2 FUNCTIONAL COMMUNICATION MEASURE: COGNITIVE COMMUNICATION Level Level Level Level Level Level Level 0 1 Unable to test No meaningful cognitive communication Cognitive communication is limited to brief episodes of appropriateness with minimal cueing; client is unaware of deficits Cognitive communication is functional only for selected activities with supervision. Client is aware of deficits with cueing. Cognitive communication is functional only in a structured environment; supervision and/or cueing is necessary for activities of daily living needs and simple ideas. Cognitive communication is functional for activities of daily living, but limited in complexity; self-monitoring and use of compensatory strategies are inconsistent. Cognitive communication is functional in most situations, although minimal difficulty may occur,, e.g., complex problem solving, latency of response. Self-monitoring/self-correcting are present. Cognitive communication is normal in all situations. (Larkins, 1987) l>——l 32 Table 3. Description of subjecte for Experiment I. Subjects : 1 2 4 Age : 41 24 29 29 Sex : F M M M Post i 1 year 6;3 1;10 12;7 injury : years years years CT/EEG : left cerebral normal none : temporal atrophic available Cognitive: VII VI VII VI level : (RLAS) : WJPEB : 15th 19th 24th 16th Memory : %tile %tile %tile %tile Airplane 1 4/2 4/0 4/3 2/1 List : (0/30 min) BDAEX : 0 3 0 2 (* Indicates the number of subtests below the cut-off scores for normal performance) El; mui V11 wh in im in it St be 33 Experimental Design and Procedures The experimental design was an alterating treatment multiple baseline across subjects single-subject design. With this type of design, at least two independent variables (e.g., intervention approaches) are compared to determine which is more effective for changing behavior. To control for order effects, the treatments are administered in an immediately counterbalanced, or alternating fashion (Kazden & Hartmann, 1978; McReynolds & Kearns, 1983). The independent variables for this study consisted of two intervention approaches for the treatment of the subjects’recall of personal names: a) a direct instruction based approach, and b) a mnemonic based approach. The multiple baserate feature of the design serves to document that any observed changes in behavior noted are the result of the experimental intervention; in that the behavior of interest should change only for that subject who is receiving intervention. Materials The materials consisted of a total of 40 color 35mm photographs, two each of 20 various adults whom the subject did not know. Contrived last names were constructed and randomly assigned to each of the 20 individuals. The last names were developed so that they consisted of two syllables, and that for each of the names, the first 34 syllable or entire word was easily represented by a visual mnemonic. For example, the name Wyner was selected because of its possibility of being represented with a bottle of wine. (See Appendix A for the names used). The titles of Mr. and Ms. were used with the last names. The first set of photographs were full-face 3/4 poses of each individual. In the second set of photographs, each individual was shown in the same pose, while holding an object that was to serve to represent the last name. The object served as a visual mnemonic to assist the subject in associating the person’s name to the mnemonic. For instance, the fictitious person Mr. Hatfield was photographed holding a hat. Likewise, Ms. Glassner was photographed holding a drinking glass, Ms. Keyston, holding a ring of keys, and so on, for all 20 individuals. Pre-test and Baseline Prior to the initial baseline session, each subject was shown the 20 plain photographs in random order, and asked, What is this person’s name?". Since the individuals in the photographs were unfamiliar to the subjects, it was expected that they would not be able to provide any of the names. After a subject’s failure to give the person’s name, the investigator provided the name of the individual, e.g., "This is Mr. Hatfield". Following a five-minute break the photographs were once again presented to the subject to assess recall of the personal names. This pre-test 35 procedure was conducted on two separate occasions within one week for each subject. On the second occasion the subjects were once again told the names. Table 4 shows the results of this pretesting for the subjects. To determine how many names that non-brain-damaged individuals would be able to recall, given identical procedures, six normal adults were told the names of the people in the 20 photographs. Following a five—minute delay, they too, were asked to recall the names. Results revealed that on the first occasion their performance ranged from 1 to 6 names correctly recalled. On the second occasion, these individuals were able to correctly recall at least five names, with the range being between 5 - 10 names (see Table 5). 36 Table 4. Pretest results for CHI subjects’ ability to recall personal names Time 1 Time 2 SI O 1 82 0 0 83 O 0 S4 0 0 37 Table 5. Results of name recall for non-brain-damaged adults Time 1 Time 2 SI 2 5 82 4 5 83 1 7 S4 4 7 S5 3 10 86 6 10 XI ll 0) [\J NI ll ‘3 (A) 38 Baseline sessions immediately followed the second pretesting for the CHI subjects. For the baseline, the 20 photographs were randomly presented, and the subject was asked, "What is his/her name?". For Subjects 1 and 3, three baseline sessions were conducted within five days, with the criterion for each subject being that no more than three out of ten of the personal names could be recalled. The multiple baseline for Subjects 2 and 4 was continued until Subjects 1 and 3, respectively, had demonstrated appreciable gains in the recall of the names under either treatment approach, (i.e., minimum recall of five out of ten items). The multiple baseline sessions for the Subject 2 and 4 were conducted three times per week. A plus/minus scoring system was used throughout the entire experiment. To receive a plus score the subject had to produce the name exactly correct. For example, if the subject said, Ms. Glasser for Ms. Glassner or £2; Lockens for Mr. Lockett, no credit was given. Treatment Treatment sessions were conducted 4 to 5 times a week, in a private room with the experimenter sitting across from the individual subject. Half of the names (ten) were randomly assigned to a direct instruction approach, while the other half (ten) were assigned to a mnemonic based instruction approach. There were equal numbers of males and 39 females in each stimulus set. Each training session was divided into two phases: a) intervention and b) recall. In the first phase, the subjects received the two intervention methods (e.g., mnemonic based instruction, or direct instruction) in an immediately counterbalanced order throughout the experiment. Prior to each training session the subjects were told that they were going to be seeing pictures of the investigator’s friends and that they were to try to remember their names. During the second phase each subject’s recall of the training items was assessed. Direct instruction. During direct instruction, each actual photograph was shown to the subject. The subject was told that, ”This is (name of person)"., while the photograph remained for approximately 3 seconds. This instructional sentence was stated only once. To avoid the effects of rehearsal, the subject was told not to repeat the person’s name. The experimenter then proceeded to the next photograph. As with the first photograph, the subject was informed that, " This is --". This type of direct instruction occurred for each of the ten photographs in this set. During direct instruction, providing the personal name for each individual in the photograph was the only information given to the subject. Once direct instruction was provided for each of the ten photographs, the photographs were randomized and the procedure was repeated a second time. Hence, for each session each subject was 40 presented with the stimuli twice, for a total of 20 direct instruction trials. Mnemonic Instruction. During mnemonic instruction, the subject was presented with both the plain photograph of the individual, and the photograph depicting of the visual mnemonic. For example, with the personal name Mr. Hatfield, the visual mnemonic for this individual was a "hat". The investigator stated, "This is Mr. Hatfield. Each time you see this (with the examiner pointing to the actual picture) picture (actual) of him, I want you to think of him holding the hat". This teaching instruction was stated once, and the subject was not allowed to respond or repeat (i.e., rehearse). Each of the ten personal names assigned to this type of instruction were taught in the same manner. This same procedure was immediately repeated after instruction of the tenth stimulus word. Consequently, each subject was presented with the stimulus items twice during mnemonic instruction, for a total of 20 mnemonic instruction trials per session. As with direct instruction, training stimuli were presented in random order. After the first phase of each intervention session, the subject was engaged in a mental manipulation task, such as counting backward from 100 aloud. In addition, the subjects were engaged in conversation relative to the day’s events, for 5 minutes. This was done to ruleout the possibility of rehearsal. In LA I“ 41 Recall Procedure. The recall phase of each session immediately follwed the five minutes of unrelated activities mentioned in the preceding. In the recall phase of each session the investigator presented each of the 20 plain photographs of the stimulus items in random order (i.e., ten from direct instruction and ten from mnemonic-based treatment), and the subject was asked, "What is his/her name?". The investigator noted the correctness of the subject's responses, keeping a separate record of those items trained under direct instruction versus those trained via mnemonic instruction. No positive reinforcement was used throughout treatment. Subject 2 remained in baseline until Subject 1 responded correctly to a minimum of five out of ten of the stimuli in either the direct instruction or mnemonic-based instruction. The training sets and order of intervention were counterbalanced with Subject 2. That is, the set of personal names trained first via direct instruction for Subject 1 was now trained with the mnemonic approach, followed by training of the first set of personal names. Except for the necessary counterbalancing, the procedure and training conditions remained the same as they were with Subject 1. Additionally, Subject 3’s training replicated Subject 1, and Subject 4’s training, that of Subject 2. 42 Reliability One unbiased judge recorded the subjects responses while reviewing an audiotape or a videotape of sessions, once weekly, throughout treatment, and compared her responses with those of the experimenter. The judge scored the accuracy of the subjects’ abilities to remember the personal names, during the recall phase of each treatment session. Reliability agreement was 99.5%, which was calculated as agreements divided by disagreements plus agreements, multiplied by 100. Results Figures 1 and 2 depict the number of names recalled during baseline sessions, and the recall phase of each training session for the four subjects. Pretest data are contained in Table 4. Results of pretesting and baseline measures clearly demonstrate that the subjects had difficulty in recalling the personal names employed. As can be seen in Table 4, only one subject was able to provide one of the names during pretesting in which the names were introduced. As also can be seen, in Figures 1 and 2, when baseline sessions were initiated none of the subjects recalled more than one name from either stimulus set. After the third baseline session, treatment for Subject 1 was initiated for the stimulus names, while Subject 2 remained in baseline. The initial effects of treatment can be compared to Subject 1’s baseline sessions, and to Subject 2’s continued responding in baseline. With the institution of treatment, Subject 1’s recall increased to five out of ten items during the first training session for the mnemonic based instruction; this compares to an increase of one item under direct instruction. Subject 1 continued to demonstrate better performance with mnemonic instruction, although with each treatment session the difference between the two treatment approaches became less apparent, and by 43 Figure 1 (Subjects 1 and 2) [flitiAIIAnICL 3-15!an! 44 SUBJECT I Figure 1. mm u . 4 .0 J . . 1 m .... . . 6 . o c. S i 6.0 . coon-IMOQIII000000000IICCocoon.Icon-I a 0.. .0 . m c a. . .n. u . T O U; .. A OI 000 E O I... m 0 O. . . o 0.. .O. . 00. co 1 I I I I I I I I I a A W l O 1.. BASELINE 098765432I0 huwumou aunt-dz 9 I0 II 12 I3 8 SUBJECT 2 TREATNENT BASELINE a c m a m N PM.» . . 4. rd 6 a u n .0 .r u. i... .0. . C. ova. r. ...o a A 0 0987654321.. buumuou “meta! 9 IO II I2 I3 I4 IS I6 8 SESSIONS Figure 2 (Subjects 3 and 4) knit-inac'u EMIIIII 4 s SUBJECT 3 TREATMENT Figure 2. m . 4 i . C I 6 00 . 1 COO-IcouocilhlcoooIIANUHQOFIuooIIclonal m Q0OihU .00 1 I... 000 4 H «HO. '0‘ .\\ NV 1 3 co c. L 000 0 I. on 000 000 I 1 2 9" 4 It. IIIIO I. 4 III Qtt. ~|O 4....” U TI \% .0. 0|! 1 m E m I“; 0‘. 000KU J 9 W m n D A. ooh 00 c S n o I Du . I 4 2.. a 1 cc 0% L .I t 06 J 7 u u v 4 m o .. 0 3% . u u * a J 6 I I I I I I I I JV . fl .0 O0 1 5 J u c 00. 0.“). i 4. M A CL I I I I I I I II I L m m 3 w A. B M A- A rIILIaIr L pl 4 T Li TL} Will: 0 [IL . L .‘IlLu'IrIanuLIIIPIIIL .0: 9 8 7 6 S 4. 3 2 I 0 m 9 8 7 6 S 4 3 2 I 0 hummus aunt... huuflua awn—:23 2 3 45 6 78 9IOIII2I3I‘IISI6I7' SESSIONS 0 46 the tenth training session she was recalling items in both mnemonic instruction and direct instruction with 100% accuracy. Following her successful treatment, Subject 1 was followed-up once a week for three weeks with only the recall phase (i.e., no treatment) to determine if she could maintain a similar level of recall performance following training. It was found that her recall of personal names receiving the mnemonic based instruction ranged from nine out of ten during the first follow-up session, but increased to 100% for both the second- and third- week follow~up sessions. She also demonstrated continued high levels of recall of those names for which she received direct instruction (see Figure 1). Subject 2 was held in multiple baseline until Subject 1 had attained at least 50% recall in one of the instructional modes. As shown in Figure 1, Subject 2 demonstrated consistently low recall throughout the multiple baseline. However, his performance during treatment was sporadic, and he never recalled more than three names in any one condition. Initially, Subject 2 was making slow but continuous gains with mnemonic instruction as opposed to direct instruction (see sessions five through seven in Figure 1). However, during the eighth training session his 47 recall for those items receiving direct instruction was slightly better than his recall of mnemonic based instruction items. During the ninth session he performed equally poor with both the mnemonic instruction and direct instruction approach, having only one correct recall from each. During the tenth through the thirteenth session, Subject 2’s recall was only slightly better as a result of direct instruction. During the twelfth through the sixteenth session Subject 2 could consistently only recall a total of three personal names for both conditions. It should be noted that Subject 2’s overall consistent decrease in performance from sessions twelve through sixteen were consistent with his having had an increase in his medication at that time. No follow-up data were collected for Subject 2. In a manner similar to Subjects 1 and 2, Subjects 3 and 4 were treated in a multiple baseline across subjects (see Figure 2). During the pretests and baseline evaluations Subject 3 could not recall any of the names assigned to either instructional mode. However, when treatment commenced, his performance increased to three out of ten correct recall in mnemonic instruction and one out of ten correct recall of those names receiving direct instruction. By the fifth session he was recalling five out of ten names under the mnemonic instruction approach, compared to two names receiving direct instruction intervention. By the 48 tenth session, Subject 3 was recalling nine out ten names correct with mnemonic instruction. Although there was a drastic drop-off in recall across eleven and twelve. He continued to make steady gains with the mnemonic based intervention, and during two out of the last three training sessions (i.e., sessions thirteen through fifteen) continued to recall nine out of ten names correctly with this approach. Subject 3’s decline in recall across sessions eleven and twelve may be attributed to some personal problems he was encountering at that time. Subject 3’s best recall with direct instruction was six out of ten names correctly, which was achieved during session ten. From the eleventh through fifteenth training session his performance decreased, ranging from three to five items correct. During the last three out of four training sessions (i.e., sessions twelve through fifteen) he consistently recalled four out of the ten personal names receiving this type of instruction (See Figure 2). As with Subject 1, one week after treatment (i.e., session sixteen) ceased his recall of the names without the training phase was assessed. Although his performance had decreased, he continued to perform better with those names which received the mnemonic intervention approach, recalling six out of ten names. His follow-up recall of items receiving the direct instruction intervention was two out of ten correctly recalled (see Figure 2). 49 Subject 4 was held in baseline until Subject 3 had demonstrated at least 50% recall under at least one of the intervention approaches (like Subject 1, that condition was the mnemonic based intervention). As shown in Figure 2, Subject 4 demonstrated continued low recall during the multiple baseline sessions. During the first six training sessions (i.e., sessions six through eleven) Subject 4 never correctly recalled more than two items receiving either the mnemonic based instruction or direct instruction intervention. It was not until the twelfth session that Subject 4 demonstrated an increase in performance. From the thirteenth through the seventeenth session there was a noticeable difference in Subject 4’s recall of the the mnemonic based intervention versus the direct instruction training items, with him demonstrating better recall of those names receiving the direct instruction treatment. In summary, Subject 4 did not demonstrate improved recall until after several training sessions. He also was better able to recall those names trained via direct instruction. Experiment II Given the results of Experiment I, particularly the unclear performance of Subjects 2 and 4, it was decided to conduct a second experiment. As with Experiment I, the second experiment sought to investigate the differential benefits of a direct instruction or mnemonic based instruction approach for the recall of personal names. It differed from the first experiment in that it was a single- subject multiple baseline design across behaviors, wherein each individual subject specifically serves as his or her own control. This is in contrast with Experiment I, wherein a multiple baseline across subjects design was employed. It was believed that a multiple baseline across behaviors design as in Experiment II provided for more control over subject variables observed in Experiment I (e.g., medication, assessed cognitive/linguistic level, time post injury). For example, during Experiment I, it was found that Subject 2’s medication regime was markedly different from that of Subject 1 (who was currently taking 3.75 mg. of Tranxene daily, to reduce anxiety, i.e., a tranquilizer), and likewise for Subject 3 (who was currently on no medication) and Subject 4 (who was taking 25 mg. of Mellaril a day to reduce anxiety, i.e., a tranquilizer). 50 51 In addition, Experiment II was motivated to assess what effects the number of training items might have on a subject’s recall performance. Recall that during Experiment I, each subject was presented with 10 direct instruction and 10 mnemonic instruction items, for a total of 20, for each training session. This was felt to be an overwhelming number of training items for these subjects, particularly Subjects 2 and 4. Given this concern regarding the number of training items, Subjects 1 and 3’s gains in treatment were quite remarkable. Nonetheless, it was of interest to explore the effects of the treatment protocol when a fewer number of training items were introduced at one time. Method Subjects Two adults, who sustained a CHI served as subjects in Experiment II. They will be referred to as Subject 5 and 6. Subject 5 was a 23 year old male who sustained a blow to the head, resulting in a CHI, 3 months prior to his participation in the study. At the time of his injury he was not employed. His medication regime consisted of 200 mg. of Tegratol daily for seizures. Subject 6 was a 30 year old female, who was employed as a waitress at the time of her accident. She sustained a CHI eight months previous to her participation in the research, as a result of her being struck by a car. Her medication for the first eight 52 treatment sessions consisted of 1400 mg. of Tegratol and 1000 mg. of Depakote, both to control seizures. During the ninth through seventeenth sessions the amount of Tegratol precribed was reduced to 1000 mg., with the amount of Depakote remaining unchanged. As with the first four subjects of Experiment I, these two had no history of previous head injuries or other neurologically based disorders, and no history of alcoholism, as determined by case history interviews. Subjects 5 and 6 were both judged to be functioning at a cognitive level VI/VII RLAS, or at a level 4/5 according to the Functional Communication Measure - Cognitive Communication (Larkins, 1987). The subjects’ language abilities were assessed with the Boston Diagnostic Aphasia Examination (BDAE)(Goodglass & Kaplan, 1983) (see Appendix C for raw scores and percentiles specific subtests). In evaluating the subjects’ performance results, the cut-off scores for normal performance and the percentile ratings based on the aphasic population were used. Subject 5 fell below the cut-off score in the following areas (see Table 6 for the total number of subtests that fell below cut—off scores): For Commands, he obtained a raw score of 9 (cut-off score = 13), ranking at the 45th percentile; a raw score of 20, for Responsive Naming (cut-off score = 27), ranking at the 70th percentile, Oral Sentence Reading, a raw score of 7 (cut-off score = 8) 53 ranking at the 80th percentile, a raw score of 8 for Repetition of Words (cut-off score = 9) and 1, for repetition of Low Probability Sentences (cut-off score = 6), ranking at the 50th percentile, and a raw score of 9, for writing Sentences to Dictation (cut-off score = 10), ranking at the 93 percentile (see Appendix H for Subject 5’s individual Subtest Summary Profile). Subject 6 fell below the cut-off score for normal performance in the following areas (see Table 6 for the total number of subtests that fell below cut-off scores): She obtained a raw score of 7 (cut-off score = 8) for her auditory comprehension of Complex Ideational Material, ranking at the 65th percentile, a raw score of 6 (cut-off score = 8) for Oral Sentence Reading, ranking at the 78th percentile, a raw score of 5 (cut—off score = 6) for repetition of Low Probability Sentences, a 3 for Comprehension of Oral Spelling (cut-off score = 6), and a raw score of 6 (cut-off score = 7) Reading Sentences and Paragraphs, ranking at the 70th peercentile. (See Appendix C for raw scores and percentiles for specific subtests, and Appendix I for the subject 6’s individual Summary Profile Sheet). Overall, no specific aphasia syndromes were observed for either Subject 5 or Subject 6. Also, when using the Bye§_to assess the areas of Paraphasia and Recitation, Singing, and Rhythm, neither of the subjects’ performance revealed any deviant behaviors. 54 The Woodcock-Johnson Psycho-Educational Battery (Woodcock & Johnson, 1977) Memory Cluster subtest was also given to these subjects. For both, their performance was at the less than 1 percentile rank. (See Table 6). When given the Airplane List (Crovitz, 1979), both subjects were able to recall 6 out of the ten names in the story immediately. After the 30 minute delay, both subjects recalled four of the words from the passage (see Table 6). In addition, CT scan information revealed that Subject 5 had left parietal damage. Subject 6 reported that after she sustained her CHI, a (frontal) lobotomy was performed, which was confirmed by her caregivers. Although, no radiological information was available. Table 6 presents the descriptive information for the two subjects. The two subjects were screened for prosopagnosia in the same fashion as the subjects in Experiment I, and they also passed with 100% accuracy. Furthermore, the subjects’ also passed a hearing screening test at 25dB HTL, at 250, 500, 1000, 2000, 4000, and 8000 Hz. Inquiries were made regarding the subjects’ wearing of eyeglasses. Both subjects stated that they did not wear eyeglasses. 55 Table 6. Description of subjects for Experiment II. Subjects I 5 6 Age I 23 30 Sex I M F Post I 3 8 injury I months months CT/EEG I left parietal none available Cognitive: VI/VII VI/VII level I (RLAS) I WJPEB I <1 %tile (1 %tile Memory I Airplane I 6/4 6/4 List I (0/30 min) BDAEX I 6 5 (* Indicates the number of subtests below the cut-off score for normal performance) 56 Experimental Design and Procedures The experimental design for Experiment II was a single- subject alternating treatment multiple baseline across behaviors design. The alternating treatment aspect of the design was the same as in Experiment I, in that two independent variables (e.g., intervention approaches) were compared to determine the relative effectiveness of each for improving recall. The treatments were administered in an immediately counterbalanced, or alternating manner to control for order effects (Kazden & Hartmann, 1978; McReynolds & Kearns, 1983). The intervention variables were the same as in Experiment I (i.e., a) a direct instruction approach, and b) a mnemonic based approach). Material The materials consisted of the same 40 color 35 mm photographs used in Experiment I. Recall that 20 of the photographs depicted a full-faced 3/4 view of individual people, while the second set of photographs consisted of the same 20 people in similar poses, except for the presence of an object serving as a visual mnemonic for the person’s name. 57 Pre-test and Baseline Pretesting and baseline evaluations were conducted in the same manner as Experiment I. Pretesting results were such that neither subject correctly recalled more than two names (see Table 7). When the recall of Subjects 5 and 6 were compared to that of the non-brain-damaged individuals reported in Experiment I (See Table 5), it was apparent that these subjects demonstrated difficulty in the recall of personal names. Baseline sessions immediately followed the second pretesting for Subjects 5 and 6. For the baseline measures, the 20 plain photographs were randomly presented, and the subject was asked, "What is his/her name?". For both Subject 1 and Subject 2, three baseline sessions were conducted within five days of one another, with the criterion being that, for each subject, no more than 30% of the personal names could be recalled. As in Experiment I, a plus/minus scoring system was used throughout the entire experiment. Treatment Treatment consisted of 5 sessions a week. It was conducted in a private room with the examiner sitting across from the individual subject. Of the 20 names, half of the names were randomly assigned to a direct instruction 58 Table 7. Pretest recall results for Subjects 5 and 6. Pretest Recall Results for CH1 Subjects Time 1 Time 2 81 O 2 S2 1 1 59 approach, while the other half were assigned to a mnemonic based instruction approach. For purposes of the multiple baserate across behaviors design, those ten names randomly assigned to the direct instruction approach were further divided, with five of them being randomly assigned to initial intervention and five being assigned to the multiple baseline. In a like manner, those ten names assigned to the mnemonic based instruction approach were further divided into sets of five; one set for initial intervention and one set for the multiple baseline. Each training session was divided into two phases, a) intervention and b) recall. Just as in Experiment 1, during the first phase, the individual subjects received the two intervention methods (e.g., mnemonic based instruction, or direct instruction) in an immediately counterbalanced order throughout the experiment. Prior to each training session the subjects were told that they were going to be seeing pictures of the investigator’s friends and that they were to try to remember their names. During the second phase each subject’s recall of the training items was assessed. The randomization that occurred to determine which set of ten names would be trained first was such that for Subject 5, the names he correctly recalled during baseline sessions were trained second. This was done to observe the subject’s recall performance when starting with a 0 baseline score (see Figure 3). To accomodate counterbalancing, 60 Subject 6 was first trained with those ten personal names held in baseline for Subject 1, which were the Set 2 names. Subject 6’s recall of the Set 2 names during baseline sessions revealed a total of no more than a total of one name being correctly recalled (See Figure 4). Direct instruction in Experiment II remained the same as in in Experiment I except that now fewer items were being trained at once. That is, with this Experiment only five items were trained initially, as compared to all ten names being trained at once in Experiment I. The mnemonic instruction procedure also remained unchanged from Experiment I, except for the fact that fewer items were trained at once, as explained in the preceding for direct instruction. During the recall phase of each session the investigator presented each of the ten plain photographs of the stimulus items in random order (i.e., five from direct instruction and five from mnemonic-based treatment), and the subject was asked, ”What is his/her name?”. Baseline probes were done each session for both subjects to assess if a change in baserate was occuring. The items held in baseline were interspersed with the training items. The data collecting remained the same as in Experiment I. The Set 2 names remained in baseline until Subject 1 demonstrated recall of at least three out of the five names in either the direct instruction or mnemonic based 61 instruction approach. The training sets and order of intervention were counterbalanced with Subject 2. That is, the ten names receiving the direct instruction approach were now receiving the mnemonic based instruction intervention. Within each instructional mode, the five names that were assigned to Set 1 were now held in baseline for Subject 2, and the Set 2 names were trained first, followed by training of the Set 2 personal names. Except for the necessary counterbalancing, the procedure and training conditions remained the same as they were with Subject 1. Reliability The reliability procedure remained unchanged from Experiment I. There was 100% agreement for Experiment II. 62 Results Subject 5. As shown in Table 8, during pretesting Subject 5 was able to recall two names; and as can be seen in Figure 3, Subject 5 was not able to recall any of the names in baseline for which intervention was first introduced. His maximum recall of those items held in the multiple baserate was two for the names to be trained under the direct instruction approach and one, for those names to be trained with mnemonic instruction (see bottom graph in Figure 3). After the third baseline session, treatment for Subject 5 was initiated for the first set of names to be trained, with the second set of names being held in baseline. Initially, for Subject 5 during the first training session for Set 1 names there was no change from baseline in the recall for the five names receiving either mnemonic instruction or for the five receiving the direct instruction approach. During the fifth training session he increased his recall of names receiving mnemonic instruction to two out of five and increased his recall of direct instruction items to one out of five. Continued improvement was Figure 3 (Subject 5) 63 SUBJECT S Figure 3. GEN TREATI‘IENT BASELINE 5%- r Ir '1‘ *"'L"OI 4. 3 2 huumfiou mum—La! .4 .A 1 I3 I4 IS I6 I7 234$6789I0|II2 O GEN TREATMENT BASELINE 0-0 0 o a I o I a 0-0-0 9' U " C I one o-c-c-a-o o' 2 mm Q. r. . . x . . O 00 . u n O .. m u will ._,. .Puwmmcu awn—L32 a. 1.. a t IIIIFIIIIA 1 4 1 J J 0 I0 II I2 I3 I4 IS I6 I? 8 9 SESSIONS 7 23456 64 noted during the third treatment session, with Subject 5 correctly recalling four of the names receiving mnemonic instruction, and three of those receiving the direct instruction based intervention. He performed equally well with both types of intervention during session eight, recalling four out of the five names for both approach. During the eleventh session, Subject 5 demonstrated a correct recall performance of five out five for those names receiving the mnemonic based instruction. He continued to recall four out of the five names receiving direct instruction. During the twelfth treatment session there was a decrease by one in his recall for those items receiving mnemonic instruction. By the fourteenth and fifteenth session, he consistently demonstrated correct recall of five out of five of those items receiving the mnemonic instruction approach. On the other hand, his performance for those items being trained under the direct instruction approach reached an asymptote, with his recall performance of four items correct remaining unchanged from the tenth training session. When Subject 5 correctly recalled at least three out of five names in either training approach for the Set 1 names, intervention was begun for the Set 2 items being held in the multiple baseline. This occurred following session six wherein he first attained a recall of four out of five 65 of the items receiving mnemonic instruction, and three out of five of the items receiving direct instruction see Figure 3). It should be noted that all the while the Set 1 items were receiving intervention, Subject 5’s recall of Set 2 items, in the multiple baseline was at a stable low level (see Figure 3). When intervention was initiated with Set 2 items, steady improvement was seen in the recall in the recall of those items receiving the direct instruction, as well as the mnemonic instruction approach (see Figure 3). During the eighth session, Subject 5 increased his recall by one name for both the direct instruction and mnemonic based instruction approach. Sessions eleven through thirteen revealed an increase in correct recall to four items for those receiving the mnemonic instruction, now exceeding his recall for the.direct instruction approach. By the final two training sessions (i.e., sessions thirteen and fourteen) his recall for the those items receiving the direct instruction approach had increase to four correct; demonstrating equal recall performance with both type of instructional modes for the Set 2 names. If one observes Subject 5’s recall performance of the entire training package, (i.e., the 20 names), by the last two sessions he was correctly recalling nine out of ten items trained with mnemonic instruction, as compared to eight out ten for those receiving the direct instruction training. Once weekly for two weeks, Subject 5 was followed-up to 66 determine if his performance would be similar when treatment was no longer instituted. As shown in Figure 3, his recall performance remained superior with those items receiving mnemonic based instruction. During both follow-up sessions he recalled all ten items receiving mnemonic instruction, and a total of eight names receiving the direct instruction. Overall, his improvement slightly changed with the mnemonic instruction approach and remained unchanged with the names receiving the direct instruction approach. Subject 6. During pretesting, Subject 6 was not able to recall more than one of the 20 names. During the baseline and multiple baselines her performance remained low. As with the previous subjects, the effect of treatment for Subject 6 can be compared to her baseline performance, as well as to her multiple baseline. To account for the counter-balancing of training stimuli, training was initiated with the Set 2 names while the Set 1 names remained in baseline. The fourth session revealed an increased recall by one to one for the mnemonic instruction names and continued to do so until the seventh session in which her performance remained the same, with three out of five names being correctly recalled. As shown in Figure 4, Subject 6’s recall for those names being Figure 4 (Subject 6) 67 SUBJECT 6 Figure 4. GEN TREATMENT BASELINE . a 0.... .n O ...O . w coca-conco-cafluaoCWoVCOIP-Idalooc cccccc i w r; 0 .u .u... .n. :6... {.0 32. Man... \O A" .....e. . m a. ; 0 no... i 8 mmmw AWN” I 7. u u .a . 16 a Q. I. I.IIIIIIH.H.I.4 O 3 2 A... W. m o .. .1 m 4. u. w. w..- 00 Puwmmou UNDZSZ GEN TREATMENT BASELINE p I. O .0 u l l DOC-OOIICIIIC0.000Q¢COOIOOIIOOIOOOCOI 6 MI .O a O... . mu... DI ..()-. b LT h h 4 3 2 I Puwmdou «metal J .1 I AAA_I\_A_A-I\ I0 II I2 I3 I4 IS I6 I7 SESSIONS 2 3 4 S 6 7 B 9 0 (h 68 trained with the mnemonic instruction approach continued to improve, as compared to those receiving the direct instruction intervention during the ninth through twelfth sessions. Furthermore, during the eleventh treatment session, she was correctly recalling all five names receiving mnemonic instruction. Despite a decrease with the mnemonic instruction during the twelfth session, she continued to outperform her recall performance demonstrated with the names receiving direct instruction, although in a marginal way. During the last two training sessions (i.e., sessions fourteen and fifteen), her correct recall performance increased to a level of four out of five names being trained with the mnemonics. Whereas her recall of those items trained via direct instruction remained at a level of three out of five. Treatment for the second set of names for Subject 6 (i.e., Set 1), commenced after session six, wherein she recalled three out of five names correct from the Set 2 names receiving the mnemonic instruction approach. Like that of Subject 5, the initial treatment session for the Set 1 names revealed no change in performance from baseline probes, which was 0 correct. Sessions eight and nine revealed equal recall performance with both types of instructional modes. Sessions ten through fifteen (except session thirteen) revealed superior recall performance with 69 those items trained with the mnemonic instruction as compared to the direct instruction items (See Figure 4). Overall, Subject 6 demonstrated superior recall with those items trained with the mnemonic instruction approach. When examining her overall recall performance for the entire treatment package, she was recalling nine out of ten names receiving the mnemonic based instruction approach, whereas, Subject 6's best recall performance for those names under the direct instruction approach was seven out of ten (i.e., session thirteen) and by the fifteenth treatment session, her recall under the direct instruction was recalling only five out of the ten names. This subject clearly demonsrated better learning with the mnemonic based instruction approach. Following treatment, Subject 6 was followed-up once weekly for two weeks to assess her recall of the personal names when no intervention had been applied. Her recall of those items receiving the mnemonic based instruction was better than for those names under the direct instruction approach. Subject 6 recalled seven out of ten names receiving the mnemonic instruction intervention compared to her recall of four out of ten names under the direct instruction intervention (see Figure 4). During the second week follow-up, she demonstrated improved recall of the personal names. Subject 6 could recall a total of eight names from the mnemonic based instruction approach and six 70 names from the direct instruction intervention. Although Subject 6’s recall performance decreased overall for both types of instruction, she continued to demonstrate better recall for those items receiving the mnemonic based instruction treatment. Discussion In general this research demonstrated that both a direct instruction and a mnemonic based instruction approach can have an impact on the personal name recall abilities of some patients with a CHI. Two out of the four subjects of Experiment I demonstrated improved name recall with mnemonic instruction, while the remaining two subjects demonstrated no marked improvement in name recall under either instructional mode. Additionally, the two subjects of Experiment II demonstrated relatively better recall of names which received mnemonic instruction; albiet a marginal relative effect. During the initial period of intervention, Subjects 1 and 3 demonstrated better recall of those names receiving mnemonic instruction as opposed to those receiving direct instruction. However, at the conclusion of training, Subject 1 was recalling items in both mnemonic and direct instruction conditions equally well. Subject 3 demonstrated consistently better recall with the mnemonic based instruction than the direct instruction approach throughout the treatment. As will be presented more specifically later, when examining the types of errors made by Subjects 1 and 3, it was found that the majority of their errors on those items 71 72 receiving the mnemonic instruction did contain the mnemonic itself, although the entire name was not correct. For example, Subject 1 was observed to initially refer to ”Ms. Keyston" as "Ms. Keyson". Likewise, Subject 3, was noted to refer to "Ms. Bellant" and "Ms. Bellum". Under the scoring procedure employed, these types of responses were considered incorrect. Neither Subjects 2 nor 4 demonstrated marked improvement in name recall for either instructional mode. These subjects were judged to be functioning at a cognitive level VI (RLAS) (level 4 (FCM)), indicating that both had more severe cognitive deficits than Subjects 1 and 3. The assessed cognitive performance of Subjects 1 and 3 was at level VII (RLAS) (level 5 (FCM)). Furthermore, both Subjects 2 and 4 were several years post onset (i.e., Subject 2, 6;3 years post injury, Subject 4, 12;7 years post injury), whereas neither Subjects 1 nor 3 were more than 1:10 years post injury. Perhaps such mnemonic instruction as a type of memory strategy yields better performance with CHI individuals who are functioning cognitively at or above a level VII (RLAS) (level 5 (FCM)), or even a level VI/VII (RLAS) (level 4/5 (FCM)) such as Subjects 5 and 6. However, Massenberg & Casby (1988) found that CHI individuals functioning cognitively at a level VI (RLAS) did perform well with mnemonic instruction when they were less than two years post injury. 73 Due to the heterogeneity observed with Subjects 1 - 4 relative to years post injury, and assessed cognitive functioning, it was decided that instead of an alternating treatment single-subject multiple baseline across subjects design, an alternating treatment single subject multiple baseline across behaviors design would better address these dissimilarities. Hence, Experiment 11 utilized the latter design. In addition, with Experiment II, the number of items trained at one time was reduced, since the number of training items presented during Experiment I (i.e., 20) was thought to present more of a problem for Subjects 2 and 4. It was recognized that with the smaller number of training items introduced at one time, there would be less room for the subjects to demonstrate learning, and as such, the results of Experiment II should be interpreted with some degree of caution. Both Subjects 5 and 6 demonstrated overall gains in recall of names when treatment was introduced, notwith- standing the smaller number of items trained. Both subjects demonstrated a trend for better recall of personal names receiving the mnemonic based instruction as compared to those receiving the direct instruction approach. By the last two training sessions, Subject 5 was recalling nine out of ten items receiving the mnemonic instruction (combined results for Set 1 and Set 2). The only name incorrectly 74 recalled from those items receiving the mnemonic instruction was Mr. Beerson, whom Subject 5 generally referred to as Mr. Beepster. This demonstrated that in fact he did utilize the mnemonic (i.e., a can of beer) for that personal name. Interestingly, during the follow-up sessions for Subject 5, this name was correctly recalled. Subject 6 also demonstrated better recall of items receiving the mnemonic based instruction as opposed to the direct instruction. When considering the total recall of items, Subject 6 was recalling nine out of ten items correct that received the mnemonic instruction, during the last two training sessions. She correctly recalled seven, and then five out of ten of those items receiving direct instruction during the last two training sessions, respectively. During follow-up she recalled seven and then eight of the combined sets of training names correctly that received the mnemonic instruction, compared to four, followed by six items of the combined direct instruction training sets correctly recalled. Subject 6 demonstrated similar types of errors during recall of the mnemonic instruction items as Subjects 1, 3 and 5, in that the portion of the name represented by the mnemonic was correctly recalled, although the entire name was not produced correctly. For example, early in treatment, the subject referred to Ms. Wiggins as Ms. Wiggy. It was also noted, that this subject correctly recalled Mr. 75 Lockett from the second through the ninth training session. However, the remaining three sessions she referred to him as Mr. Lockens. It was interesting however, that during the one- and two~week follow-up sessions, she correctly recalled this name. When examining the types of recall errors made by Subjects 1, 3, 5 and 6, 91% of the errors made contained the correct mnemonic when recalling those items receiving mnemonic instruction, although the entire name was not correct (e.g., Ms. Combreak for Ms. Combrink). As mentioned previously, these type of responses were considered as incorrect under the scoring system used. Had a multidimensional scoring system been employed wherein partial credit was given for certain responses, a higher score would have been yielded for these subjects. Errors made during direct instruction did not have such a mnemonic aspect. Interestingly, only 12% of the errors which occurred during direct instruction could be similarly interpreted as mnemonic based. Also, the recall attempts of the two subjects who demonstrated low gains in either mnemonic instruction or direct instruction, very seldom, if ever, demonstrated a mnemonic bases. This type of error analysis, in concert with Subjects 1, 3, 5, and 6’s relative improved recall of mnemonic based items, demonstrates that these subjects did indeed incorporate the visual mnemonic strategy into their attempts (correct and incorrect) at 76 recalling names receiving mnemonic based intervention. Reviewing the data from all six of the subjects, it appear that those subjects judged to be functioning at cognitive level VI/VII to VII (RLAS) (Level 4/5 to a Level 5 (FCM)) demonstrated the most gains from either the mnemonic based instruction or the direct instruction. When comparing the relative effects of the mnemonic instruction to that of the direct instruction approach, the data seem to demonstrate a trend that mnemonic based intervention leads to better recall performance. The population of CHI is very heterogeneous with regards to age of onset, site and extent of lesions, following neuropsychological deficits, as well as times since injury and initiation of a particular intervention approach. The subjects in Experiment I and II certainly reflect this hetergeneity with respect to these areas. As was found in Experiments I and 11, not all subjects perform similarly relative to treatment. However, four subjects (i.e., Subjects 1, 3, 5, and 6) demonstrated improved name recall relative to baseline performance and relative to multiple baselines. Additionally, there appears to be a relative effect of mnemonic based instruction over that of a direct instruction approach. This research examined the effects of mnemonic instruction versus direct instruction on the recall of personal names in CHI subjects. With four subjects (i.e., 77 Subjects 1, 3, 5, and 6) there was a trend for their recall of personal names to be better when trained with a mnemonic based instruction. Two subjects (i.e., Subjects 2 and 4) did not demonstrate marked gains with either instructional mode. Subjects 2 and 4’s reduced cognitive functioning (i.e., level VI, RLAS, (level 4, FCM)) paired with the amount of time post injury (i.e., 6;3 years and 12;7 years, respectively) may account for their reduced learning ability. 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APPENDICES Appendix A Personal Names Females 1. Ms. Wyner 2. Ms. Lightsie 3. Ms. Keyston 4. Ms. Rosen 5. Ms. Lacy 6. Ms. Bellant 7. Ms. Glassner 8. Ms. Penrod 9. Ms. Wiggins 10 Ms. Combrink Males 1 Mr. Hatfield 2 Mr. Booker 3. Mr. Shuman 4. Mr. Beerson 5. Mr. Appleby 6. Mr. Roper 7. Mr. Spooner 8. Mr. Lockett 9. Mr. Nutkins 10. Mr. Hartwell 88 (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding (holding APPENDIX A a a wine bottle) lightbulb) ring of keys) CREE) piece of lace) Dell) drinkingjglass) an ink pen) a a a a wig) can of beer) an apple) a a a rope) §P__00_9.). combination 1995) an walnut) (holding a paper heart) Appendix B (percentile and raw score/maximum score) 89 APPENDIX B Performance on the BDAE SUBJECTS SubtestsI 1 I 2 I 3 I 4 Fluency I I I I AR I 70 (7/7) I 70 (7/7) I 70 (7/7) I 70 (7/7) PL I 70 (7/7) I 70 (7/7) I 70 (7/7) I 70 (7/7) ML I 60 (7/7) I 60 (7/7) I 60 (7/7) I 60 (7/7) VA I 80 (13/14): 75 (12/14):100 (14/14): 80 (13/14) Aud. Cp I I I I WD I 100 (72/72): 70 (64/72): 100 (72/72): 100 (72/72) B-PI I 100 (20/20): 100 (20/20): 100 (20/20): 100 (20/20) Com I 90 (15/15): 90 (15/15): 90 (15/15): 90 (15/15) CIM : 70 (8/12) I 80 (9/12) : 85 (lo/12): 90 (ll/12) Naming RN CN AN 0. Rdg WR OSR Repet RW HP LP Auto. Sp AS Rec. 100 100 99 90 100 90 90 100 90 80 (30/30): (114/ : 114): (22/23): (30/30): (10/10): (10/10): (8/8) : (8/8) : (8/8) : (2/2) : 100 94 91 90 100 90 80 90 70 8O 90 (30/30): (109/ I 114): (ll/23): (30/30): (10/10): (10/10): (7/8) I (6/8) I (7/8) : (2/2) : 100 100 100 90 100 90 90 95 90 80 (30/30): (114/ : 114): (34/23): (30/30): (10/10): (10/10): (8/8) : (7/8) : (8/8) : (2/2) : 100 100 91 90 90 70 90 85 70 80 (30/30) (114/ 114) (13/23) (30/30) (9/10) (9/10) (8/8) (5/8) (7/8) (2/2) 91 Rdg. Cp. I I I I SD I 70 (10/10): 70 (10/10): 70 (10/10): 70 (10/10) WR I 80 (8/8) I 80 (8/8) I 80 (8/8) I 80 (8/8) COS : 100 (8/8) I 75 (5/8) I 100 (8/8) I 100 (8/8) W-PM I 80 (10/10): 60 (9/10) I 80 (10/10): 80 (10/10) RSP I 100 (10/10): 80 (7/10) I 95 (9/10) I 70 (6/10) Writg. I I I I Mech. I 90 (5/5) I 90 (5/5) I 90 (5/5) : 60 (4/5) SW : 100 (47/47): 100 (47/47): 100 (47/47): 100 (47/47) P-LD I 90 (15/15): 90 (15/15): 90 (15/15): 90 (15/15) SP-D I 100 (10/10): 85 (6/10) I 90 (7/10) I 90 (7/10) WCN I 100 (10/10): 100 (lo/10): 100 (10/10): 100 (10/10) SD I 100 (12/12): 100 (12/12): 100 (12/12): 95 (10/12) NW I 100 (5/5) I 90 (4/5) I 100 (5/5) : 90 (4/5) Key: Fluency-- AR = articulation rating, PL = phase length, ML = melodic line, VA = verbal agility; Auditory Comprehension-- WD = word discrimination, B-PI = body-part identification, Com. = commands, CIM = complex ideational material; Naming-- RN = responsive naming, CN = 92 confrontation naming, AN = animal naming; Oral Reading-- VR = word reading, OSR = oral sentence reading; Repetition-- RV = repetition of words, HP = high probability, LP = low probability; Automatic Speech-- AS = automatized sequences, Rec. = reciting; Reading Comprehension—- SD = symbol discrimination, VR = word recognition, COS = comprehension of oral spelling, V-PM = word-picture matching, RSP = reading sentences and paragraphs; Vriting—- Mech. = mechanics, SV serial writing, P-LD = primer-level dictation, SP-D spelling to dictation, WCN = written confrontation naming; SD = sentences to dictation, NW = narrative writing. Appendix C APPENDIX C Performance on the BDAE (percentile and raw score/maximum score) SUBJECTS SubtestsI 6 Fluency I I AR I 50 (6/7) I 70 (7/7) PL I 50 (5/7) I 70 <7/7) ML I 50 (6/7) I 60 <7/7) VA I 50 (8/14) I 80 (13/14) Aud. Cp I I VD I 100 (72/72) I 100 {72/72) B-PI I 100 (20/20) I 100 <20/20) Com I 45 (9/15) I 90 (15/15) CIM I 90 (ll/12> I 65 <7/12> 94 Naming I I RN I 70 (20/30) I 100 <30/30) ON I 100 (114) I 100 (114) AH I 91 <12/23) I 92 (14/23) ORdg I I VR I 78 (24/30) I 83 (27/30) OSR I 80 (7/10) I 78 (6/10) Repet I I RV I 50 (8/10) I 70 (9/10) HP I 75 (6/8) I 90 (8/8) LP I 50 (1/8) I 85 (5/8) Auto. Sp I I AS I 70 (7/8) I 70 <7/8) Rec. I 80 (2/2) I 80 (2/2) 95 Rdg. Cp. I SD I 70 (10/10) 70 (10/10) VR I 80 (8/8) 80 (8/8) COS I 100 (8/8) 60 (3/8) W-PM I 80 (10/10) 80 (10/10) RSP I 80 (7/10) 70 (6/10) Vritg. I Mech. I 90 (5/5) 90 <5/5) SW I 100 {47/47) 100 {47/47) P—LD I 90 (15/15) 90 (15/15) SP-D I 85 (6/10) 85 <6/10) WCN I 100 (10/10) 85 <8/10) SD I 93 (9/12) 99 (ll/12) NV I 80 <3/5) 80 (3/5) Key: Fluency-- AR — articulation rating, PL = phase length, ML = melodic line, VA = verbal agility; Auditory Conprehension-- VD identification, Com. material; word discrimination, = commands, laning-- RN = responsive naming, B-PI = body-part CN complex ideational 96 confrontation naming, AN = animal naming; Oral Reading-- WR = word reading, OSR = oral sentence reading; Repetition-- RW = repetition of words, HP = high probability, LP = low probability; Automatic Speech-— AS = automatized sequences, Rec. = reciting; Reading Comprehensione- SD = symbol discrimination, WR = word recognition, COS = comprehension of oral spelling, W-PM = word-picture matching, RSP = reading sentences and paragraphs; Vriting-- Mech. = mechanics, SW = serial writing, P-LD = primer-level dictation, SP-D spelling to dictation, WCN = written confrontation naming; SD = sentences to dictation, NW = narrative writing. Appendix D 97 SUBIEST SUMMARY PROFILE NAME: Subject 1 DATE OF EXAM: PERCENTILES: 0 IO 20 30 40 SO 60 70 80 90 100 SEVERITY RAIINC o I 2 I I o ARIICULAIION RAIING I 2 4 S 6 Q . PHRASI them I I 4 ' o IIUENCI ’ /‘ MEIOOIC UN! I .' I b . VIRBAI «CIIIIY ' o 3 a o a “N I4 wow DISCRIMINAIION 0 IS 25 I7 46 5! so e4 67 70 AUDHORY com-mun IOEMIIICAIION o I 5 I0 I I :5 l6 I7 Ia COMPREHENSION commwos o 4 o 5 I0 I I I I I4 . COMPux ID€AIIO~AI MAYERIAL 0 2 I 4 s 6 g -/ II I2 RESPONSIVE NAMINC o I 5 I0 IS 20 2c 27 2 NAMINC CONFRONIAHON ~w~c o 9 25 4 I so 72 a4 94 I05 ANIMAI. ummc o I : I I 6 9 0/2) wono READING o I I 7 I; n 26 ORAL READING om SENIENCE READING o I z 4 7 9 atmmo~ 06 woaos o 2 s 7 a 9 9 REPUITIO\ MICHI’RIIBABIIIII .I I 3 I I ‘ ‘. uMJ'IIoII \aum I) : 3 4 o \‘ \(IIIIIIJsm‘ «I In a 4 .‘ ' O ' . IIIIRAI a: I' :: 9 o. 3 I 2 I\ 0 PKRAPHANA VERBAL 40 23 III IS I.‘ 9 7 4 I I \. txn~o¢o 75 I2 5 I I o AUYOMAIIHO Si (NCIS 0 I 2 I 4 6 7 AUTOMAIIC SPEECH 0” athtmc o I swam OIscammAnm o 2 s 7 a 9 K NIT I ' 7 RE ADI N C woao ucoc On: 0 I I I e O\ COMPRIHUHSION 0‘ ORAL SPtluNc o I 4 o 7 O COMPREHENSION // WORD-POOL!!! MAICHINC 0 I 4 3 9 .. IIIAOINC SEVIINCES mo PARAGRAPHS o I 2 I 4 s b 7 \K 9 Manama I 2 3 I 9 sum wmmc o 7 Is 25 I0 II :0 u «I PflIMfl-ltvu OICIAIION o I 4 6 9 II II N <‘ WRIHNC SPIIIINC. IO OICIAII0~ n I : I 3 r ' WRIIIIN C(NIRUMAIION VAMINC o I 3 I h ° 9 D Shawls m OICHIIm. o I I a a ‘ VARRAIIVI wanna u I : I 4 . \IL‘SIC >I~(.I.~c u I I RHYYHM O I \C' Appendix E SEVERITY RATING FlUE NCY AUOIIORY COMPREHENSION NAMINC ORAL READING REPUIIION PARAPHASIA IUIOMIIIIC SPEECH RE ADWC COMPRLHLNSION MINING MUSIC . 98 SUBTESI SUMMARY PROFILE NAME: Subject 2 DATE OF EXAM: PERCENTILES: 0 I0 20 30 40 SO 60 70 80 90 I00 0 I 2 I 4 O ARIICULAIION RAIINC. I 2 4 s e I PHRASE IINGIH I I 4 I o MELOOIC LINE I 2 4 o O