mm UEL Uxmmm PLACE IN RETURN BOX to remove this checkout from your record. TO AVOID FINES return on or before date due. MAY BE RECALLED with earlier due date if requested. DATE DUE DATE DUE DATE DUE 2/05 cz/cTicxoatm.md—ma TILE-.3) :F‘L'I..;'LII’TETC Iii ULi'Jj‘Cf. CHILL i‘a-I‘i G L A I: 0 125x91“. 3 UL) ENC-0,3! 7 197 7 'Ei'iTrJSI'u‘VI Oi" C(JIITTIT’L‘S USE-"'8 I. 111;”){LAT-)EJCTIOI‘IOOOOO0.00.00...OOOOOOOOOOOOOOIOOOOC II 0 O1". LL'Z"JIL‘J'¢. (2F I'llf‘lj‘J 1:. {L313IJJ‘LLE'I‘ZO . C O O O C O O O O I I O C O O O C O O O O . CAUSES or uNIInuooo LEAD PCIGQhIEG.......... ETFbCDS or CHILDHOOD LEAD POISONIKU......... 1» DEE—“:73?“Z'IL'i'i AZTD ‘1'.{5"2311‘1-,I'AI‘IT Of" CI'EII'DTKIL‘Z’ TEA?) .' ' ‘n -\u - I OWL») i-’ D filly-\Ioooooooooooooo00000000000000.0000 2O ’7'} f. 5"! x r: p- Ill. 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I) IA FECTJUTSS jg, 1-13 LI 00 .1111” 2' EY Firure ~r‘ ° 1‘ l {TUITC Firure Fixfllre Fi6urc Fi'uro Firwrre Pirure Ti6uro 1. ’) l.- -J LIEHP 0F 13 IDES ixoo6 nre of YounF Philczren to Environment? 1 Fen' anfi 016 Urban mousinfi' 3:636), in Rosoirrtory Exoosure to Tera :oflecfied in_the ican Blood-L936 Vflues of Vfrious Grouns Lech Paint Regulations - 25 SESAS With the Greatest Iurber of LBLS Current Programs 113T OF FiGUdEU Seasonal Distribution b6 .6nth of Kenort of Fftai :66 Eon‘atal C3686 of Ienfi Poisoning an6 Increasefl Lead Absorntion Iumbcrc of 06666 for htch Pi.6r06tic C] 8831- iiCGtion, browned by ”mflhs of £66 at Time of Disz1osis: fitnosnhoric Concentrcti orn of 1666 From Ve- hicles from miO-iSb? thr016h 616—1069 alon6 flifferent sections of U 3. 16h 96 1 in New Jer6ey ‘re6icted Nationwifle Incifcoce of lea6 Posed fuirt noiconinr - 2' 31336 With Create6 t Pre- 6ict66 Inciéenc i L c- i" H. C”) H (w ‘ .- t‘. ‘A . .‘I ‘ ,‘ 'fi . u Census Tracts shake: LC or6116 City of low York - fienorted 0:6 -.8 of Lead Poi— 6,016. i 11:6 Soot 16h of P10 Case6 of Y666 Fcisor -166 by PhiFFdanhia {@31606606 of ‘6t161‘s; 1056—1060 Childhood toad Poihoniu6 Control Trorraw St‘fi— J- oer 101") CaU666 ¢n6 Conse"uerccn of Chiifihnod 3086 ioisonin6 966 0'66 it] Sites 'or Intervcn— tion I11 ’1‘ £011 UC 'I' I OH ...cities have shown 6 uninue and endurir6 nro— nensity to create health nroblems end then elect to treat the symntoms rather than deal with the causes. Alonso S. Yerby, h.3. One of the host serious urban health nroblems evident in our cities today, is that of lead noisoninn. A health nroblem of many dimensions, lead noiscnins orininetes from a variety of sources and affects a varietv of victims. In children, at least, it is also Wholly nreventuble. The substance of concern — lead, is an element which oc- curs neturally in the environment as less than one her cent of the material in the earth's crust. Lend is defined as a nure metal and is used cs such for the manufacture of plumbing nines, as C‘J n additive to pesoline, nesticides, end tcirts, in the ”ro- cessing of many metallic and chemical nroducts and in the ma- kine of nottery.l In usins lead, mun tends to overload the environment, nar- ticularly the urban environment. Tons of venorized lead are emitted into the atmosnhere daily by 6onstant auto traffic. Meny structures ere neinted internally ind externally with lead- based nuints. Pottery containing lead is found in mfny urban households, and various oceunetions utilise le'd as e raw ma- terial. Conseouently, lead can enter the huzrn boéy in several ways - breathir6 nolluted Cir, drinkinp centemineted water, 7 . eating lend—exnosed foods or eatin6 :rcm hirh lead dishes, cc— 1. ’) cunational exnosure, and insestion of lead-based naint.‘ Al— though in recent years, snecific concern has been raised with recard to lead emitted by the use of alkyl antiknock compounds in gasoline, when the total environment is considered, two to three tiJes as much lead is added from saint ninments and other lead nroducts as from the use of lead alkyls.3 It can be seen then, that the eroblem of lead noisonins is indeed extensive, bervasive, and comnlex. This never will deal primarily with the most nrominent and serious manifestation of this important health hazard - lead noisonins in urban chil- dren, resultjnr from the ingesticn of lead-based taint. The effects of airborne lead a a comnlicatina factor will also be considered, but the basic thesis of this nener is the brimacy of leaded saint as the cause of childhood lead noisonins in urban children. In the first section, the causes, ccnseouenccs, detection and treatment of childhood lead noisonins will be described. Section II discusses legislative annroaches to the nroblem, nresents some investigative modelins technisues and a brief examination of the exneriences of several cities which have attempted to deal with childhood lead n7i6onin6. Secticn III presents some snecific recommendations, followed by Section IV the concludinr section. 9 O‘JE-E‘JIEI'I 0]“ THE: ~P:{(')BLEI‘-'I CAUSESOF CHILDHOOD LEAD FOISONIKG lead poisonina in children, resulting mostly from ingestion of chins of lead-containing taint from walls and woodwork in old, dila— pidated housing, remains a unioue nublic health rroblem. Its etiolosy, nathoaenesis, nathoohysioloay, and enidemioloay are known. Practical methods are available for scree- nina, diagnosis, nrevention, and treatment. Yet each year lead posionina continues to cause the deaths of many children and men- tal retardation or other neurolosiesl han— dicans in many other children.4 Lead poisoning is not uncommon in the United States. While the precise incidence of lead poisonins is not known, surveys have shown that 10 to 25 per cent of young children living in deteriorated urban slum housing shcw evidence of increased lead absorption, and 2 to 5 per cent show evidence of poisoning.5 Aporoximately 30 million housing units built before World War II are still in use today. As many as 7 mil- lion houeins units are deteriorated and contain surfaces cov— ered with lead naint, and it is estimated the 2,500,000 chil- dren live in substandard housing where a notential lead hazard exists.6 Taken together, these statistics yield a frinhtening potential. Sluntarcas in the larrer older cit es seem to have the areatest incidence of lead roisonine, concentrated in their so-called "lead belts." The nroblem seems to arise from the interaction of two rrimary causative factors: nica and the environment. Tie term nica, can be defined as the habitual, 3. purposeful and compulsive search for and ingestion of nonfood items such as clay, blaster, laundry stfrch, ashes, putty, string, naint chins, naper, dirt, crayons, yarn, matches, and cigarette butts. Adults as well as children may develon nica and both are usually selective in the materials they ingest. Studies have shown that nutritional deficiency is not an etio- logic factor in urban slum children, and in fact 50 per cent of the children from both middle class and lower socioeconomic groups habitually engree in pica. Pica usually benins at one year of ase and disannears between the apes of 3 and 5. Nearly 50 per cent of mothers_of children with nica, have nica them- selves, and the child with nica freouently stimulates the be- havior in younger siblinas. The interaction between_child and mother is usually the critical factor in the develonment of nica in children. Sianificant interactive factors may include: an absent or poorly functionine mother, or an emotionally trou- bled-mother. In such families, pica often noes unobserved by the mother, thereby nrecludinq early detection in the child.7 The amount of lead that is received bv the child t.rough pica is also sisnificant: A chip of naint about the sire of an adult's thumb nail can contain between 50 and 100 millisrams of lead, and so a child eating a few small chips a day easily innests 100 or more times the tolerable adult intake of the metal! 8 dtudies in selected slum ar-as reveal that 50 to 75 per cent of old houses contain dcnserous ouantities of flakine . .. c (2-... .. lead-based paints on interior suriaces.’ stealinss in these areas may have several coats of saint on walls, woodwork, and 4. ceilinrs, and the base coats srnerally contain sisnificant amounts of lead. Lead containins naint was "recuently used for both interior and exterior dwelling surfaces until about 1940. Such noueinn is usually in bad revnir end naint neelinns and loosened blaster provide a hazardous source of lead for children with pica. c‘ The usual locationsof leaded naint chewed by children de- .‘ veloninn lead noisoning are window sills, nainted nlaster, and walls. Outside sources commonly include nsinted door frames, _- 10 fences, DOTCAGS, and house walLs. A study in Cleveland clearly substfintiates this relation- ship between childhood lead noisoninp and old deteriorating urban housing. In this study, it was found that 2f ner cent of 901 nre—school children residing in old housine had absorbed ab- normal Quantities of lead and 35 (4.7 ner cent) had symptoms of lead noisonina. One hundred and five comnarable nre-schoolers living in better housing were also tested and of these, only 3 showed evidence of increased lead insestion and none showed clinical symntoms of noisoninn.ll (See Tfible 1.) Similar stu- dies have been conducted in other cities. Table l. hnvironnental hxnosurc of Younn Chil- dren to Lead in New and 01d Urban housinp location R0. of No. with ho. with of home ‘Childrffil abnormal '?lumbism Studied Urine (lead Poi— soninn) one Housins 801 216 _ 23(4.T$) New fiousine 105 3 0 Project Source: Chisolm and haplon, p. 944 In Ealtimore, QO oer cent of the renorted cases of lead noisonine are in children who reside in multinle—dwelling, rented housinc units. Su -rvevs in Baltimore, as well as in Philadelphia and London, iave revealed that 70 to 30 ner cent of interior neinted surfaces contain more than 1 nor cent lead. L xtraoolation of a limited survey in Boston, indicated a 98 oer nt nroba bilitv of houses with positive findinas for lead if a greater number of samnles had been analyzed. (Investigating actual cases usually reouires analysis of 20 to 95 different samnles in ordzr to identify all interior euri'ee. nositive for lead.) Geosrachic snot maps have been Rent by the Baltimore Uity Health Denartnent durine the last 30 years and indicate that as old inner—city dwellines are renlaced by urban renewal and other new construction, thelocation of renorted cases of ad noisoninr moves outnard from the center of the city and that cases are now foun -d in 0' er hous ins in nearly all tarts of the city.-2 As stated earlier, lead noisoninn in children is believed to be due almost entirely to tr e reoetitive eatinn oi leaded on retro- CL house naint. Mort of the available data are brse snective analyses of . mall Frouns of cas-s from cities in which childhood lead noisonine is a reoorta ble disease and in which the municinal health departments have active rrograms such as New York Citv, Chicano, Philadelnhia, and .Elaltimore. While the nat‘onw de incidence of the dis e~as e in children is unknown, the followine nine coiderio We ical f: ctcrs are well—establshed: 7 Areas of "hi”h risk" are nrimarily centered in the slums as LCCe”"‘bljle to fl: kins naint and broken elaster, hieh incidence of ,,iCCs., and lack of adeouato "arertal sun- ervision nrovide En ootimum environment for lead nelsonina. 6. 2. while children between 1 and o years of are are the lain victims, 85 ner cent of the cases are children from 1 to 3 years old; more than 50 ner cent of the deaths caused by lead poisoning occur in 2 year olds. 3. Childhood 1ead noisoninc is sirnificantly related to nica, as 70 to 90 ner cent of lead— noise oned children exhi- bit a history of this habit. 4. Symntometic lead noisonine in children ve r:ies by sea— son and there is evidence that 80 to 85 per cent of the cases occur in the surmer months of June tiroup h Sentem- ber. However, more cases are being renorted in the winter months as awarene"s of the nroblem amons health Workers increases. (Some winter cases occur when leaded battery cases are burned for fuel and the fumes inhaled, or there is nrolonred contact with the asies ) ”hile lead encenh- alOpathy (clinica 11 lead poisoning or brain injury) is more freouent in the summer, asynntoma tic (sub—clinical) lead noisonins is a year—round disease. 5. Ne," .roes and Tuerto Ricans exhibit a hirh incidence annarently because a nreater nronortion live in "lead belts” and not because of any known penetic, ethnic, or racial factors. 0. There is no sisnifica=nt difference in incidence by sex. 7. A hish incidence occurs amonr siblinss wiih a 30 ner cent incidence rate cited bvs01e investiertors. c. There is a high recurr nce ra ate. 9. Lezd noie onins associated with nice is a chronic ero- cess and from 3 to 6 months of fairly steady inrestion is required in most cases before clinical svnntoms apnear. 13 (See Fieures l and 2.) Estimates of the- incidence of lead nois chine and the nre- sence of adverse blood level concentrations are same on shaky foundations. AS stated treviuoslV, the dis mrse is only renor- table in a few of the iarscr citi s. Usually, reco,nition is "sed on the case— fincins annroach, which is dencndent on the level of local medical community awareness and the aveil: bility of diagnostic laboratory facilities. Prosnective rass screening nrogrcms are currently available only in Chicano (sin nce C-cto— - . -- . -. , . .1, 14 , ber 1906) and in LOW York City \s1nce January lt/U.) Although 7. Firure l..3¢f 30 '28 26 24 NUMBERS OF SUBJECTS M f JAN. Nonfatal Source: f, O 48 ’- 44 ~— 40 - w h- 8 321- V) d U 28 _ U. 0 24’ 2“ ‘ U (D :5 20»— D Z 16 L- 12 »— 8 >- 4 1... Fisure Groaned 13/4 C T11_T-11_1 D Asymplomulc mace-rd lead abmvpuon E Lead pouch-m) I i .19: Anne lead encophdwpamy. muld ['3 Acme lead encephahnuhy, some 9 Dulh L buyer at. l-’.-,- EL_MJMD_1_ JUNE . 111111 ._.....J DEC. Wtal lead Absorntio 28 26 24 72 2O 18 16 216 FEB MARCH APWL MAY NOV. MONTHS onal 1st r1_but ion by Vonth o” eicrt of 08309 of 1-1. Poisoninv and Increased omnittee on iriiolosic Lf:{ects of AtmOsnheric lollut'nts, .5 ourc e : U. 136 1-mfiriamma11n_1.--i_n _____ n 111111 :2. 011 364] 1859 6071 I283 (193 961W AGE AT TIME 0‘ DEAGNOSIS K‘MOUPED BY MON "1.3) P. anhcrs cf Cvses i1r L"ch bi: nost.ic Clfis by Font‘s of see it “time of Uicsros is. Go ..... ittee on mioloric 4ffects of htnosnheric E] Ltad wusomug Dram ~~-—.-un -‘ i“ '1 ..', .. I. P 2 Asymmnmahc Invent-«4 ti ad ab'uxphu'n V Iii-j AC "a lead enccr.-‘\a|"1:.nhv. mm! E] Arutr Bead oncvuh fluoa'hy, sown! L...“ - m- 103 119 1D...___ “3013! 68 44 40 .ritication, “ ‘ .Lr‘ J- .1'0 '11). ml". 1. r3 “’9 adequdte enidemiolosical data 01 the distribution of blood lead concentrations in children are lareely unavailable, larae—scale screening programs in urban areas sueaest: ...nerhans 5 to 10 oer cent of youns children who live in deterioratine old housing have con— centrations of lead in their blood that may be associated with adverse metabolic effects, at the very least, and 1 to 2 oer cent mry have evidence of lead noisoninp. where active screening programs are in oneration, the num- bers of renorted cases increase, while the number of reported deaths decrease to well under 1 Der cent of renorted cases.15 In addition to lead~based naint itself as a causative factor, several other factors aearavate and naenify the nro- blem. One such factor is the lack of awareness about the pro- blem aiona nhysicians and other health workers. Many physicians are unaware of the existence or the magnitude of childhood lead noisonine, because they seldom encounter cases of it, or be- cause the cases they do encounter are-incorrectly diaenoscd. Another common misconceotion is that the manufacture of lead— free taint today has resulted in the extinction of lead poison— ina in children, without reeard to the fact that old houses often still contain many layers of lead—based he nt, paints man— ufactured for outdoor use still contain lead, and such outdoor paints may be used for interior surfaces by uninforxed neonle. Those Dhysicians and health wcrkers who are aware of the nroblem of lead poisoning, are often hesitant to make a nositive diag- nosis, resultina in undue delays in treatment and the loss of . lo valuable time. A poorly informed nuhlic is the second cornlicatinp fac— tor, as many narents are unaware of the danrer associated with nica, or tre consecuences of taint intestion. AdditionallY, 8. inadequate srevention of reeXsssure to lead results in a hinh rate of recurrence of lead noisoning amonp children. The fail- ure to nrevent reexnosure to lead contributes sreatly to the mor— tality and morbidity of lead noisonins. In addition to noor housins conditions end the lack of awareness on the nart of nrofessionalsand the nublic, inadequate health and housins codes also contribute to the nersistence of lead ooisonins. In those cities with codes snecifically nro- hibitine lead naint in the interior of dwellinss, enforcement of such codes is usually unsatisfactory for reasons such as eXpense to landlords, the city‘s fear of abandonment by land- lords, a shortaae of nerscnnel to carry out the necessary en- forcement nrocedures, and difficulties and confusion in legal nrocedures and concepts. As lin—Fu states: Failure to pet rid of lead naint in a house where a child is kno n to have develoned lead noisoninn usually means that a treated child returns to the same hazardous environ- ment to be execsed to another enisode of noisonina. "ven if the afflicted child is moved to another heuse, the bro- blem still remains: If the leed nflint in.thehouse is not removed, the lethal heritase will soon bass on to other families with children, and lead noisonina amanr children multiplies.18 although the slum areas of lures urban centers exhibit the highest incidence of childhood lead noisonins, the arc- blem is not restricted to poor slum dwellers, as cases of lead 1.1 *1 {'0 :3 Q4 poisoning have been resorted in children from COCiTlH economically advantased homes. Urban migration tatterns from central core.cities to the suburbs have correlated well with an \0 o increase in cases of lead noisonina in nreviously unrerorted areas, as older housina is subdivided into anartments. In Baltimore, 48 mar cent, and in Philadelnhia, 50 ner cent of .— the rerorted casrs of Lead nosionins amona children occurred . . . . 19 out31de the inner Cities. dural areas and small towns are not immune to the oroblem either: Of 230 rural children 1 to 5 years old, tested in New York's Dutchess County and Connecticut's Litchfield County, 9 ner cent had blood levels considered unduly high. Some of these children were from unner- and middle-income families. 20 In a recent study of 0,500 children in lfl Illinois cities with ponulations raneine from 10,000 to 150,000 nersons, 18.6 oer cent carried hish levels of lead in their blood. (300 micro- arams is the daily permissible intake fronifood and other sources.)21 Still another previously unrecoeniaed notentiel lead noisoning-causina situation, is that associ{ted with tte re- modeling of old homes. Horkmen as well as children End their oarrnts can set lead noisonins simnly by breathirs in finflll nerticles of lead durina the nroeess of rewodeljns and renova— tinr inner city houses with.lead-based weints on interior sur— faces. In a study by Wolf, of schrCl cases in the Urnitol Hill area of dashinaton D.C., the residue of the heavy sandinp and soraninr in houses underaoinn renodelins was tested. It was found to have a hirh lead content. Wolf does rot surrest that neonle ston rerodelins old hous s, but does recommend that certain rrecautinns be taken to aviod lead neisoninn inclu- ding: Wearine face masks, mains a vet men after cleanina us 10. debris, not eatins it the meme room where work is ”cine on and insurinr that slates and utensils are not exeosed to lead dust, 22 q and having. body leao levels checked neriodicclly. From the noint of View of contamination of the total en- vironnent with lead, lead-usina indnstries, taint niements, and metallic nroducts 11: vs each contributed more than burned leaded. fuel. Thus any eronosal to rid the environment of lead nollu— tion throuph the removal of lead from autor.otive fueis must 9 take into consideration the existence of other lead nroducts H c‘.‘ '9 *3 L? 0 such es raints and manufactured The veneral nioture that ewerees from con— s'derction of lead is the environment is of a steep sraCient oi nollution erovat1ns rem tee cities in nronortion to their size. file tbsence of si niijcant unnard trenrs in t e concentration of lead in rural soils and water and in the food sources of ran in the last 30 years indicates that the nreat in— crease in the combustion of lead slkyls has not had 0 lzrce iILrnc ct on the intrke of lead by nonurbfln neonle and animals, excert ner- Iaes by direct inhalation.23 h“: t-h ') The magnitude of transfer of airborne lead to the soil is direct— ly rel: ted to the isity of auto trf fiic. In urban areas, tn, ,- 0 surface soil of marks, treet dust, and narrow bands beside ma- \ jor roadways are heavily contaminated with lead. Only in tre ur‘ocn settine is mzn no sibly o‘nl'ec to h”7'roou circumstances relative to atmosnheric lsM nollution, ocounaticnal exnosures in the lead-usin" ind stries ex— cented. The hier concentration of lead in urbtn air end on the surfaces of nrrks and streets constitute a source of intake add— itionfl to t e usual dietary sources find in sneci: l circ Us races may be a substfntial source.24 Animal :‘uerage—Microzrgms cf Lead Per Cubic Meter at A?! K! m 7 ,000 Vet icles Per 03 ‘3' h: m, - y ' ”I! R" .3 Wm " Mum.‘arm "aw "ma “,0 200 3’0 40") C100 he! fmrn Highway Firure 3. Atmosnheric Concentrations of L,ad Trcm Ve- hicles irom mid—1967 throuch mid—l?o9 along different sections of U.3. His'v 83 l in N w Jersey. Source: Grain and Berlin n. 6 while the reneral urban nonulation faces no identifiable current threat from the concentration of lead in the air, tn'o groups of re0ple are exnosed to airborne lead to what seems to be an undesireable degree. The first sroun is those persons who are exnosed to unx> .3 ually hip:h concentrvtions of lead in ambient air far occunationol reasons — "'rrce \orkers, traffic nolice, and lead trades workers. Such exnosure can nroduce blood lead concentrations in excess of AO/qp; tqp=microgrcms and refers to micrograms ner 100 milliliters of whole blood.) How— ever, this level of blood lead concentration is nrobably only attained by a relatively small nronorticn of those exnosed.25 The other aroun significantly threatened by lead in E sbient air consists of infants End small children. decent survers of chil- 12. dren in laree cities indicate that manv had blood lead concen— trations in the ranses of no to 604‘s. while such hieh blood lead concentrations cannot be cefinitelr said to arise from the inhalation of lead, it is likely that at least some of the lead burden in children does come from the ingestion of lead—bearins street dust and soil, which freouently reaches lead concentra— . . n 26; m, . tions in excess of 20004h/g. \see llee 2., Table 2. Resniratory Exnosure to lead fieflected in the mean Blood—head Values of Various Groups POPULATION EXPOSURE MEAN BLOOD (nicxoeiiis Phi LHED (11030- CUBIC phThx or this rte AIR) 100 GiflhS) 16 21 rural U.S. 0.5 l O downtown Thiladelnhia 2.4 2A 1 8 urban U.S. Cincinnati policemen 2. 25 Cincinnati traffic 2. 30 nolicemen Los Anneles traffic 5.2 21 nolicemen Boston Automobile 6.3 30 Tunnel emnloyees Source: Chisolm, n. 21 the study which attemnted to determine the extent to which the innestion of dirt and dust contaminated with lead exhiust from cars’contributes to the childhood lead nroblem was con- ducted bv Hear and Aronow. while most lead in dirt annears to be a result of deteriorating lead—based house saint, Hear and Aronow utilized a tracer nrocess to determine the amount of lead a Child tight receive from eatins dust centerinated with lead sources other than paint chins (nrimarily leeded arsoline.) The tracer, Le d-2lO, is naturally occurrirn end is nresent in 13. relatively ltree amounts in dust and “articulates, but nearly absent in raint. The results of the study showed that children with nica and other evidence of hieh lead intrke, end norncl children both excreted identical awmunts of Lead—210. Thus, this study concluded that dust and air—swsnended narticulates were not the sources of lead in these urban children.27 Another study, however, concluded that st eet dust was in fact a notential source of ineested lead, nerticularly for chil- dren. In this study, monthly dustfall semnles were collected in 77 midwestern cities in less. Che mean lead concentr tion in dust from various sect rs in each city was calculated after averagina the results for all of the cities in this way: rcsidcntial.....l636 s/s of dust commercial......24l3 s/s of dust industrial. . . . . .15124' {tr/gr. of dust It was determined from this study that "the swallowing of as little as lfih of such dust could result in the oral intake of an amount of lead that exceeds by e factor of l? or more the estimated mean daily intake of lead from normal fcod and . . . . 28 drink in non-eXrosed chlldren." ‘ .other =tudy, conducted in 1071, dentine lead levels ’3 '5 In ,3 I. were measured from the shed deciduous teeth of children in two Philadelphia school districts. One district, ristrict S, was considered ”hiph risk” as it was within the acknowledscd "lead belt" of the city. The second district, District 3, was con- ,sidered low risk. District 5 was characterised by a nredominantly knack nonulation, which while mobile, tended to stay within the district. The houses were older than 40 y are and many were in a severe state of deterioration. The western half of district 5 is the Ucknowledsed "1 ad belt" of the city, from which many 14. ca see of lea ad noisoninr are resorted. The eas:ern nortion of the district is hiahly industrialized with a nredo ominantly white nonulation. Housin: is old, but in generally hood re- pair. Diagnosed lead noisonins is are from this Tr a. Nistrict 8 is the area into which lhiladelnhia exncnded efte er World War II. {ouses are newer and in seed reneir. The nonulation is nredominently white. Dimsnosed lead noisonine is extremely rare in this district. In all, lol children with no frior histvry of lead poison— ins' "ere tested for dentine lead 1 Hvel The resul,s of the study sho‘ed th t blacle children in nublie schools from areas :3 "J J of det eriorc“ ted housins, had mark ed elev tions of dent inc le W1 th 20 ner cent of the children havinp levels in the range as— sociated with toxicity. In general, the white children in the newer housine had the levest levels, but as an unexnected result, a group of white children from intact housing, living near and attenli ns school :djscent to a sajor lead nroces sor also had elevated dentine lead. Because of this nroximity to a major lea d processor and also because of xtresely heavy auto traffic in the area, the authors concluded thet for thee e chi dren, airborne lead was an 1H”O rtant vehic of exnosure. In their discussion, the authors stc te that their study demonstrates that exnesure to lead noisoning is more widesnrced than had been reverted, and also suseests sources of lesd other than saint. Furthermore, nonulatiwns other than these trLditionally acknow- ledged are heine e"nos ed to eiolori celly in.nortant amounts of lead. 'ihe iindina of elev;ted lead level.» in urban tflack children livine in deteriorated housinfl 15. and in white ehildrer whose hovcine is vener— ally in Food reeair, eusnests thrt both oaint and airborne lead are onerative factors, and that children in deteriorated hourino the live in areas of heavv vehicular traffic flow are in fret beinc exnosed to both sources.29 while it has been shown thzt contaminated street dust alone does not account for clinical lead noisonine in children, the swallowin: of such dust may sienificantly account fer the higher mean blood lead content in urban children and the relatively laree fraction hevine a blood lead content of £0 to 60/«E. Com- bined with nica for paint, an increased intake of lead from con- taminated dust would result in a total load intake sufficient 30 to cause simntomvtic illness. However the direct investion 9 of lead—oirnent naints is unmistakeably the nrincinel environ— mental source in cases of severe acute lead noisonine in chil- dren.31 Other studies have attemeted to determine whether persons (not necessarily children) in urban communities show an in- creased absoretion of lead as comrared to non—urban coauunities. In 1 61, Hofreutcr studied the blood lead concentrations of grouns of oeovle from 6 American cities and comoared them with those of a rural nonulation. Taking a number of factors into consideration such as sex, and smokin; habits, Hofreuter and his colleeeues were able to show thct for each sub—aroun, urban lead concentrations were in excess of rural values. In 1065, husbaum attempted to determine Whether body burden of lead cor- related with the lennth of tin snent in a city (Los Anecles) b” studying bone snecimens. He found no correlation, however, between bone lead concentrations and “ennth of stay 12 the area. Another study conducted in 1903—71, determined the concen- 15. tratien of lead in the ambient atmosnherc at we semnlina sites in 8 American communities. (This study was an cxeansion of and a follow-um to a similar study carried out in 1962, entitled "Survey of Lead in the Atmosnhcre of Three Cities.”) The nur- nose of the study was to examine the extent to which the blood lead levels of selected nonulation erouos reflect cxoosure to lead at various levels in community atmosnhere. The study was conducted in 3 reaions reflecting various reoeraphical and cli- matological Characteristics and included the communities of: Cincinnati, Fhiladelnhia, Les Aneeles, New Yo k City, metroooli- tan dashinaton R.C., Chicano, Houston, and Les slamos (N.T.). In order to insure as s ecific and consistent a relationshin to known air levels of leal as possible, the non lations studied were nrimarily women volunteers livine within a nrescribed reaion surroundina an air samnline instrument. Whe study was concerned exclusively with tbsorotion of ambient cornunity atmosnheric lead throunh the resniratory system and did not involve nedia— tric exnosures or oceunationally—associated exnesures of ren. The study indicated that while urban levels of blood lead are hisher than suburban levels, air concent~ations of lead are not clearly reflected in tte blood lead levels on a rene‘al nctional basis. Thus, there are factors other than atmosnheric lead level which are of relatively greater imnortance in 'eterminina the blood lead levels in nonulation arouns. The erecise nature of these other variables anneared to differ teens the regions stu— died and was not defired. Between reniors, lead from food in- take is believed to br sirnificant 5nd elirete may also be im— portant, but it urban—suburban comnfrirons, tirherne lead levels seen” to be the rajor deter inent. this study did not attemet III. . . . i - 3 to examine the relatimnshin between are are blooe lead level. The association of “ica with childhood lead noisonins was ‘14. first nostulated by duddock in 1924.“ hesnite the variety of evidence and often conflictina data, it is nrobably safe to conclude that inaestien of lead—based naint is still the chief source of lead noisonire in children, with inhalation of air- borne source aearavatinn existine symntoms, but nrobably not initiating them in the absence of lead—saint nica. EFFLCTS OF UHILBHOOD LEAD lulSvKlRG It has been known for over 100 years thtt lead is an aborti- facient and that female lead workers had a his} rate of miscar- riepe. Paternal actors have also been known to be sicnificent for many years. It annears that a man sufierina from lead noi— sonina may nass abnormal snerms to a woman, resultirc in chil- dren born with lethal malformations. Even those children who anncar to be normal, may carry an elevated concentration of lead from birth, and thus may be more suscentible to further contamination from any source.35 Children who are born normal and later develen lead ooisen— inn, are likely to be left with hermenent nhysieloeical and men- tal damage. lead poisoning is cumulative. Symntoms beein to annear some weeks or months followine the continued ineestion of small amounts of lead. Early symntoms may include irritabil- ity, fretfulness, or disturbed rastrointestinal function, char- .aeterized by lack of arnetite, constination, vomiting or cramps. More severe intoxication results in lead encenhalitis (brain in- jury) due to intracranizl nr ssure. The acute stares of the 18. disease are manifested in Chen es in mental state, ataxia {ir— resularities in the functions of the body), nersistent vomiting, muscle weakness or naralysis, delirium, stunor, come, convul- sions, and frequently death. lead noisonins is further known to have an inhibiting effect on red blood cell develonment and may cause damage to kidneys and liver. The disease in children differs considercbly from that in adults. A ults rarely develon central nervous system complications or encenhalorathy, while eerinheral neuritis, lead line of the sums, and colic, common symptoms in adult cases, are rare in children.36 While increased efficiency of treatment has decreased the mortality rate associated with lead noisonins, meny victims of the disease suffer some form of intellectual or behavioral se- quelae toomnlications.) The ssecific sequelae seem to be deter— U1 mined by length of exnosure, sneed of diaeno is, and the method of treatment. In cases of reexnosure, the rrobebility of ner- manent brain danene is 100 per cent. The severity of intellectual sesuelae rnnears to be closely related to whether or not encephalonathy is uresent. If it is nresent, the nrobability of sequelse is at least 4“ *er cent. The snecific canabilities usually affected are nercertien, form discrimination, and lanruase skills. Ueterrinntion and dies— nosis of eroticnal and behavioral sesuelae is sore difficult and subjective than the intellectual comnlications just described as ”the effects of the syndrome are honelessly confoundrd with the effects of environmental denritation, which often character- ises the lives of children in the socio—economic area which pro- "37 q uccs the lareest incidence of lean rsisonins. Children with C); lead noisonins usually ethibit hynercctivity are easily distrac- . 7 . 19. ted ene +101 1 V labiJF. 1Fer also tend to he cnfiressive end hfive difficulty in establishinr ”normal” social relatinnshins. There a nears to be a close rclztionrhin be— tween severity of into"iceti'n and emotional seoue1”e. Also, the tvre of treatvcnt Ph- D”rcm tlv does not have a stron ” rel"tion to err;ctio1'f1 1- sesuelae, '-.'."nile the ebi111,v of the .1 relents to nrovide suwnort ooes.38 n q a 1 l . Accordine to a recent Juo_ic Veslth Jervice ienort (1071) ann 01:1..ntely 500,00 Arerican children have elevated blood lev- els and some 10,030 reeuire treatment for lead noiscnine. Annu- ally, 20? children die end COO ere so severely injured as to re- quire nernenent care. Thiry—two hundred suffer wodcrate to 0. - . - . . . . 3 severe Drain serene, xecuirin" substantial sne01elJVed care.” Thus, with sufficient evidence to indicate that lead ofn cro- {J duce disturbances in body Cnd brain metabolisr, end cause as— tinn mental imnnirment, it is essentie that ever” effort be node to reduce the body leed burden of children to 93 low a level as noseible. DETECTED? AND TRHATIEhT CF CHILDHOOD LEAD PMISCWILG Evidence of lead noisonine is best obtrined throunh.com- munity screening. rive es sentie l f:‘ ctors must be considered in blannine such a screening nronrcm: l. The children at risk — which children, by nresent kn wledec should be tested? As leaded pa ' nt her 1; ieentifies the children 0t risk, screening should ('enter on those areas where there are older homes in poor renair, hevine surfaces painted with lerded .nint. 2. iccess to the children — whet orsnn'7ations c2 n most readily reach these children? Parents are the key to reachins childr at risk, as educated 20. narents can exert effective pressure to obtuin relevewt tests for their children. Althoush several facilities may combine to I..J pool their outreech efforts, screeninfi of 81 chil ren at risk may not be feasible, and thus it is necessary to ninnoint tar— get areas.- One why of determinine such terset areas is to assure the children already identified as lead noisoned define hish risk environments. In some cities, this icy be 3 sinsle local— ized area, while in other cities, cases may occur over a larse part of the citv. In the latter situatinn, the indicator cese can be used to determine "clusters" to be screened. Such. cluster testinr could be done by the heclth deeartment upon notification from a laboratory testing for elevated blood lev— els, and would irrolve visiting the indicator child's residence to test other household members between 1 end 6 yezrs old, and neiehborhood children (where housinc conditi us WPrrPnt it.) 3. The technology of testins - whet technolosy is avail— able for doins the screening test on children? The technique of obteininn snecimens hfs been simnlified by the substitution of finrer sticks for venenunctur.s, allowins more peonle to be treined tc take snecinens. The success of any screenins nrogram is denendent on the accuracy std velidity of the laboratory date, as both netient and entircnnentnl follow- un rely on laboratory results. Because of the difficulty and exnense of creatinfi end maintainins a hish finality lab, ell_but the larrest nrcrrems Fenerally contract for the tork. djcal fol— we V 4. Follow—us — What are the sechcnisws for n 1 01‘7" L113 ? The blood levels 3t which vnrious actions are taken are some- what arbitrary 3nd vcry from nrosrem to nroertw. In Newark, N. J. a blood lead level under LOfiws, reuouires only a reheat 21. test within a year if the chil” still seems at risk. Jitt a '7 1 blood lead level between 4d ant 52/Wg, the child is considered to have evidence of increased absorntion and till be tested at l to3 month intervals until the ertent of the denser is clari- fied. If thr child exhibits a blood lead level of bQ/qg or more, hosbitalisation and tree mert are reuuired. These children who require re—testins and re-examination must be under a ohysician's care, although his services mey be augmented wit} the helm of hora-professionals. Screeninn brosrams must be eble to entici— nete the needs for various services, and the families' ways of paying for outnatient and hosnitul car. should also be consi- dered in the olrnnins nrocess. Ad itional factors to be dealt with, esnecially in urban areas, are the nroblem of missed an— nointments and the fact that children may be tegen to more than one facility for health care. each facility, #hile only incidentally aware of other services to individual nation.s, tends to assume natients lost to follow-us are receiving care elsewhere. Difficulties are further in— creased by the mobility of the urban nonulation. In a Kewark lead screenins survey, 40 ner cent of the families had been less than two years at their nresent address. For lead noisoninn, some common resistry is scential. In hewerk, this function is nerformed by the Centrel lead deeistry.40 5. Environmental control to clininete the source of lead ooisoninn is exnens ve — What ontions are there for envi— roneentel control and who will set oriorities for their annlication on an onsoine basis? Somethinn rust be done to the walls of the house in which lead noisonina has occurred, and althoueh a variety of vethods exist, they all rresent nroblems. H311 coverinrs in houses with lead . - . q the _, . .aints usually held for only a few months eve tOAWCLK infra- structure of such houses. dtiffer coverinr such as beaver board works tell but is flOTG ennersive. Pfiint :ey be flared 22. off but this involves substcnfial mechanically requires intense end tion of the child after tr _.e.tme1':t offers no nrotection to new yeuns Innovfitive fundine mertc for revolving lo: need exnloration. er ful nroerex of environr brinte leeisletion end enforce resnonsibilitv removel of the surface .‘1 I“. “A 1'! GCZ"... h.ze r“s, 1rd renovine noint costly lebor. hile reloca— is 9 tennornrv solution, it occur nts. nisms, such Cs erranee- he end t:K incentives, ereouie ite for a force— ente control is ennro- reeula tione tc fix and for reh“bilit:tion (i. e. or rrkire it inaccessible )41 Screenin’ is most ef ectively accomnlished throueh a COOn- rative effort emonr existine orfcn'r'tions end asencies end by buildine unon the health service structures elreadv in oner— étion within a community. "Centrz lization end senarement of the nroeram, 7111 almost certainly reouire a snecinl office to nrovide comxunicrtion, leadership, end access to tle fee ilities needed for the combined efforts ‘1 owerd the comnon of elim— "42 real inetine childhood leod noisonine. The innortent stens in di: nosine lead noisonins as out- lined bv the U.S. henerteent of Health, Education, and aelfar (HEJ) in a booklet entitled, ”The ecoenit n of lGLd ‘cisonine in the Child" fire: 1. ht"rere'1 that nice may be essocieted with lead ncison— inn. 2. Tresence of symntoms sue? as leek of ennetite, list- lessness, increased irrit bility, v (me 1.1670 i1r main or cremns, constinction, or vomiting. 3. Convulsioxis or unexnleined unconsciouh ss. 4- Sneciul loboratorv tests ere indicated W1en sue niciL n is nresent err include tests for the nresence of in iron 5 efici encr: C new 1.2:, ebnorrsacl the red blood cells, bones as seen bv X- C.y, rnd concentrations of lead in ebnorme blood blue enots or ”s+ irnlinr in l density of t1e ends of the 51 tests for abnormal erM’11rine.fi3 sneci The i entificatien of a child vzith lee .d noisoni1e is just the beginnine of several nreventivc and therapeutic measures. First, and most imnortantly, the child and the source of lead must be senarated, initially by hosn italization, if the child is toxic. Jhile in—natient treatment reeimens vary, all are based on chelation - the use of c11erica ls which combine with lead and thus facilitate its urina.rv excretion. In milder ca see, the chemical edathamil calcium disodium (CahDTA) is used, While in more severe ca ses the aeent is l3rit1ch hnti-Lewisite (EAL). while these drues are life—saving, the? ere also toxic.44 Lone—term care is escentie, and is the nest difficult and most imnortant eW1ect of treatment. Lone-term care is dependent ubon the nrecent that no child ever returns to a leaded house This Csrect of theraoy requires the coordinated effor;: of nublic health authorities to efiect the remova.l of hazardous lead sources, as stznc for the mother in her dues st for safe housine, and increasingly, mobilization of the conmunity it- self. heeeuete care often reouircs brief hosei- talization in a convalescent fecilitv or foster home. Thereafter, the child must be followed closely un til he reaches school are. where nos— sible, enrollment in nurserv school or Head Start nroerams is Cdvisa ble to nrovide stimulation for the child, thich may for the pre—school child, reduce his enotionel needs for rica.45 Because of the deny aedical and environmental factors in- volved, it is difficult to make a comnrehersive estie ate of total medical and related exnenses attributrble to lead noi- sonins. Generally sneakint, the tyne of exnosure, the ass of the cziticnt, and the severity,.recurrence, and seduelae of the illness are the imnortant variables influencing total direct medical costs in eases of lead noisonine. Sencration from exnosure is the ess.ntiel comnonent 24. of lead noisonine therapy and thus hosnitolization is freouently indicated. Obviously, costs frr treatment fall most heavily (ind directly on the poor, who are the least able to nay. Treat- ment costs can oe divided into six eeneral cetenories: 1. direct medical costs for acute and convelescent core 2. Cfter—ctre and excess school costs for the partially 1 brain—ccmasee 3. custodial care for the nerrfine1tlv and severely in- jured 4. correction of hazards in housins 5. nreventive hee th sunervision 6. sunnortine municinfil and state health denartm nt act- ivities It is difficult to senernlize about the economic innact of child— hood lead noisoninr as diernostic 8nd tr&£‘tmc:71t f cilities. vary Widely amone connunities 3nd few of thee e heve nrorrams Which can be considered either comnrehensive or Wholly adequate. In Baltimore, children ere treated under the senerfl medi- cal oolicy that no child found to have increased lend absorp- tion, with or Without svmntons, is returned to a ”loaded" home. The child is first trected in a eencrel hosnitzl for a brief beriod and then nluccd in a convalescent ffcility until a safe dWellinr (modern n1 1blic housins or adeduetely renaired old hous inn) is found for the ‘emily. In a F“0Uh of 45 3altimore children treated from 12e5-1e7o, the avernre ote.l tixe of acute and convalest cent ho snitulizetion was 100 drys and tte averaee r”ire-ct hosn1telifi”t1on cost for 34 natients was 2746. The two hirhest hosnitul bills were over $9000 each - in one case a res ult of rene ted hosnitslizetion for cornlicetions of encenhalonathy and in tke other cece, from fin excessively long wait for admission to nublic housins. Acymntosetic Children c basis in c The t rouehly es follows: 3. These Giro repairs to In Baltixo While New on be trertcfi at lower cost and on an out-natient onvcleecent i:cilities.r otel flirect mecical cast of lead “OiCO‘infl crn be timetefl accordins to the fin 1 clinical outcome, as osymntomntic increased lead 8 sorntion without obvi- ous resifluul oermrnent injurr: 31500—2000 her eatient moGerPte he moment brain damage (snecjml schooling requireo): $13,000 per oatient severe nermanent brain demase (institutioncl care re- quired): $245,000 her natient (1972 mcéical costs) ct tre t ent costs ecn be contrasted wit? the cost of substandcro housine to eliminate the nfint hazard. re, hose costs rinse from 5190—1700 oer acartxent, 47 York City estimstes costs at fil?b3 oer anartuent. Ageeuute housine retair is therefcre ccm arable in dollars with the direct xcfiiccl costs of trestment of a sinsle asymptomctic child with increaseo lead burden. Elso reecirc to sub— stenflerd houslnr can nrevert lead o isonins in €11 children who may live in a riven house durine the remainder of the house‘s useful ex- istence. If houses were inseected 9nd renaired before tre onset of nice, direct medical costs could be totally eliminated.48 2C). AE—‘l‘-L~.UACH;.E~5 To ,*.--:{.=.:3'r-:I;.: IJE SluiTILJN On January 13, 1971, rresioent Nixon siened the Lead—Based Paint Poisonine Prevention Act (P.L. “1-865). It 'cs the first federal law desiened to red we the k:20.rd of lcfld noisonine for the nation's children. lssehtielly the Act erovideé fisc and leeal sueeort for the followine: Title I - firents up to 75 her cent of total cost for 10031 “Tofrans, for detection end treat_eet of lead- bwsed euint noisoninr, includine community testih.” 3nd follow—ue and edUCPtionsl ororrrms outlininr he Clth dan- Hers. Title II — Grants in conjunction "ith thrs C under Title I for the elimination in rerideotiul hovm jn" of lead-based otint on interior end other surffices to Leich children may be commonly exnosed. (floth titles cull for emoloyine reeieehts of communities or neirhborhoedc to cerry out ournoses of the Act.) Title III - A federal rese?rch and oe“.onstration nrohrsm to ceternii: :e the exteifi of lead eois onine and to find more effective ways of controllins lead neint risk. Title IV — Authority to erohibit fut re use of lee5.—’r:ed etint in residential structures eittcr ccnstrrcted or re— hsbilit1ted after the effective cate of the ect.49 (See Annendix) The fit, authorired nonronriati«ns of 51 W,» 0 ,0 in l€7l and iPO,Vio,JoO in 1‘72 to carry out its erovic ions. Amendments to the :ct were mode in l”72 and l“73, With the le .tt er roue fl" } incorooretine rest of the elements of the earlier, rfmely: 1. an increase in fedcrur feedier free 75 eer cent to 93 nor cent of the cost of local “roerets 2. an exornsion of the fefieitioe of ”leod—brsed emint" from 1 her cent lend content to c.0e eer cent 27. lusion of the leC5—htsed nnint neisonine nroblcm scone of the Pub‘ic ueelth Service Act A. the creetion of e fictionel Childhood Ieee Based Paint Poisonine Board to advise HEN 5. n: ohibition of leCd— hes tures receivinn federal assi niture, cookine, drinking, and e: The ameneed Act Ceeronriates 3o1,3oo,ooe e veer for two years of thich $25,000,000 is for detection and tree newt of lead noisenine, 535,050,000 is for elimin notion of the neieonine and ::3,OJ), 000 is 10f Jeze earch. 50 Under this Act, HEJ hes nro- vided funds for (0 cities to start or exwend efferts to ere— Vent leCd-besed nnint noisonine. In addition to these 40, ennroxiznetely a dozen ot*er cities heve stfrted nroerens either in the health Henertment or the Housine Insnector's Office, or both, with many other cities indicatinr their desire to start a lead poisonine nroerun Es soon as enoueh nublic end official interest to Cllooete iujids is renereted. In e5diti(:n to fun- dine, technical heln is also available throuel a National Clearinphouse on Lead taint Poisorinp Irevention es tcblished bv Hhfl in the Health Services and ientel Health £5ministretion in Rookville, tnrylnnd. The federal eoverneent is Clso ooinr ree cCrch to imnrove and sienlify screenin", testine, and treat— ment methods.51 In fiifiition to felerfil leeislction, other hinEs 31 level solutions to the nrohlem of lead neieoninr need to be considered such as housine code reeulstion bv lo Me eovernnents and land- lord—tenant innovations. Several chIies h”ve n'eeed ordin.t:nces (ieelizwrxvith lxvui ntirn:‘noisordjvf, nleoirxr it in iflne'brocd context of eovernmentel code enforcement. (Sec TCole 3 ) PO 1") Table 3. load ”0-4-1 AO”_*—‘ Q “.‘. 3f?jfi1t i . [Abel U - health ‘ . nvt 0n . LIX-Ht I ("'V Area 01_ {or II L.|or [hutxfixxtq ‘ SLzrflnck‘s .3310 Bxltimorc Housing yefi vc< Boston chltlx yes nChicago Housing yes ”.5 Cincinnati Health yes 50» Connecticut Health, yec \vx . {musing {“nczccsible i >Ul‘(‘.¥« Pa") ! Jersey City Health 5r: ye: ’ Mussachu>ett< HL\11L \Ls yo: 0 ' t‘xffx‘ihl‘ .‘vuuarL H0311}! \, . x. . New Huvtdx Hedltia, ch )vfi + “quaing (Jexowslhle (~_\‘?{‘) in] ‘ :‘(N (‘21.:Vlllh !L”\lth \(‘.V "'f“: . (AggLfifiIan 1xh‘l'it71} XrK \Irk VYJLC HrJXIL \yu Veg ~ . (detuniblv exterinr) New kark (iitv In.311}1 f . ‘ s Nzrfoll h ”‘11. Rhodc Isluxul luqlith ‘hiluJClphid H‘ner .~‘ y.~ 3 ft. LuutS ix:.\u \\ 3w . - 'li'~v!!:[l.‘ -.',Y."{1::=;b LJ‘nington, H.C nzAlth, .(w Manning Kilrihgton ”(fillh ye~ M Source: }ilsinn, n. forujfrbiwns tilt. farce R (‘21:0V3! y.-_ .— _._.»- yvn veg \c< \'L‘. 5 u .__.._.—.._._._ ____._ bourCu _.-_ ___.,.___ —._.- Rwltzwwxp Lvrl *lStl, fi{”/33, rn1\ lu'q ., -v lJiH' Labeling 0rdin1ncc ”easing Cole, Oriinance Wnniyipul Cnie ef (Hi:\go. gcctggn 78-17.2 Cincinnati Le1d Urdirxnce, 1960 {w‘lic Act l0: An A;t to Enforce th: E11min.tan uf Lend Es$ed Faint in Housing KKZOmOJJYionV, 1971 (itv Oldir1nev ’C-ifi — fin Ordinance Repu‘ i ln'xng the W1lc Hf 'vaJ FJtnt in the City I 0f Jtrtcv City, 1963 th1ptcr 109). An fiLt Pre\1d|nq for a Cnn- yrvhvnfi} c “r(”er of Lead Poisoning Pre- xentirn unJ Cart: [-1971 Urdilmn " funnieru 3‘1113170, 35102170. °PIU£1"V, 81102270. and FFIOZI‘O of the ('ity of .‘.‘r-:..\:‘lx, N.J., 'I'N. 1370. inrndnvnt uf va.inu CaJe relative to Lend S‘nxwr (n) Hue]?irp,t‘nirs ltN-E - Lan ikllnt :ICiHJHCL' l‘|(.'l deinan\e RI“ unenlvd 19'] '{{1Li\? Vn“yil}fiUJS 31 Vodefi, Rules 3rd P: wlution. If the State if flew York _ mural) mm. 'm t3 wand the Fu-“lic‘ Hrrlth 13v il!‘h*rx’k‘H To Vb? Prevention Jul {cn‘rol ~f Lend Peiaoning ‘ Alflin‘ktrllixc {air - lend 7uint .\:lwt|nnn 1T‘0 .01 vurl l \ A "guil;i(n~ Fel”in‘ I. Labellng, fippli- .11“. And ECHOYJ’ hf IULH PLint 195“ < 9r 2 in} nu.- - I.‘.f-’ H i ".‘el ‘ .. i-=-)|n i11:! (‘l'xl 111:111c c [j] '1 H 112% ”" HIJITTH- YJlt ' U"P :§ [CH4 ! ininr, 337‘ Hun in; “.qwinvinns Sr;tinn “VJianrv 'vgn‘uIiLu wri finscrning fl qxvnr -f Hunt'r; ng In. Yvnq J ing [mic 'H “Elwingtun, D 1., “C“lth , Lead Pniwt, 1953 7 . -__. 7-. ‘--—- . . . However, a variety of nroblems arise in at c ntino to enforce such a muricirfll orfiinance. Some of these oroblems are related to the manrer in which cefie violations are Getcc ted. Voluntary renortine by occunants is inexnensive, but may not be satis— I4) ectory as eccun: nts mav refuse to disclose violftions of which they are aware. By renortinr an infraction, a tenant may bring about retaliation, eviction, concemnation Cf the building, or rent increcses. Feriodic survey insoections bv the municinel- ity are both costly and may ceus e 86ministrctive eroblems. Oc— cunants may deny citv insnectors access to units. Owners may obstruct code enforcement when the cofie is too strict end if compliance reouires ler e ex: nditures. A second tyne of oroblen associated with cooes, coxicerns the tyne of enforcement itself. Strict enforcement may compel some occunents to vacate condemnefi or (htnfloned substandard housing and seek shelter in tighter marks , whereas loose enforcement, \vhjle oossiblv maintainine the hour inr mcrlcet, may involve and~oermit the occunancy of unsafe and unsanitary units. Owner's resnonse to a municinel code rroerum is 8 third kind of rroblem. fiesnonse is Effectefi by the substvnce of the code ane tie l.oca tion on holdinrs, tax considerctions, and q overall orcfit nicture. Tonoloros are hesit rt to fenrove in herd—core slum areas where nronerty v;lues are reduced as buildines one one the area's socio—econoeic status declines. Also, if the cost of irnrovements is nascee on to the tenants via rent increases, the toner may have flifficultv firdine oc— cuernts or pres ent terunts rev vacate. leeorfiine tex consider— 2 '2? . ations, althounh removinc the load hozsrd from a dwellins unit may not be an assessable imnrov:ment, Fenerel tux ores ures on the owner mny affect his investment scheme so es '0 nreclude voluntary renoir exncnditures. Some owners may simnly be un— able to nay for buildinn imnrovene ts, and even if lone—term financing were available for improvements, mrny mieht still re— J frein believing that the buildine -oes not eerit inveStment, or the fear of poine into debt. The alternatives fecine an owner Who does not consider his returr on imnrovements to be adeouate are: continuinn oneretion in violation of the code, ellowinn the city to make the necessary renairs, or slendonnent. The final set of nroblems concerns those leeel nethods which code enforcement asencies may be authorized to use. Crim- inal brosecution is the foremost method of enforcement, with. fines serving to deter infractions and induce res irs. However, this aonroach has often been criticised. First, determining the fine is difficult as it must be lsrne enough to be considered more than a nonlinible cost of business and small enough to leave canital for the needed reneirs. Rise, mony owners may be willinn to risk Onnrehension if enforcement efforts seldom resu7t in nrosecution, or when the cost of comnlience is firea— ter than the amount of the nrobable fine. A fixed oer diem civil penalty for each dey t violation nersists miort encourese rabid rensir to avoid a lorne total fine, but many landlords might continue to risk erosycution as that nronosel would Unnenr not to increase the nueber of violations enueht and nenslised. Equi— eble remedies such Cs injunctions and receivershin ire effec— tive, but require lerre commitments from the enforcement arencies. 30. injunctions reouire the owner to rencir or cease oncrctins his buildi in", with eouity nrovidinm needed suoervision of the com— pliance effort and the sveilablility of contempt sanctions. In the receivershin orocess, hen bos ically sound buildihes are not renuired, the court—unoointed receiver mekc. renoirs and 12! ms es th: bu i1 ire until the amount of rent collected eouols H {+3 the cost of repair. Desnite the substantial enforcement efforts involved, the use of these tho methods in controline the ero- blem of lead poisonine can be desirable. n direct municipcl emergency reneir nroyrem is a fino' alternative when the owner does not re eir and immediate work is necessary for occunant wel- 52 f8 re. Jhen one considers the unwillingness of rsny owners to renuir, the inefficiency of crimi— nal sunct ions and fiscal limitations on city novernments, txe nrosoect of solvins the lead boisonins problem is poor. It has been sug- nested thet the frustrations of C code en- forcement nro"rom lead to "a tendency to ne— sotiF to the tenant's riehts sway with the hone that sooner or later e.rd o.deQUFtely financed rent—subsid3 or oublic- housirrr nrogrcm will elininzte the slums. In cons ec,uence, s vic— ious circle of non-cction is ere? ted."53 In Cddition to municinFl codes 9nd lows, the existence of e leidlord's oblieeticn to keen his tenant's nremises free of lead nuint hazards may be derived from soecific state leni- slstive enactuelt more reneral stetutorv duties, or judicial .P‘n internretetions of the com:on 1e".: . sechusetts, beryltnd, and New York have nusFed statutes which imnose a duty not to €nnl y leao o".int to the interior .urieccs of dwellinss. Thir— teen stFtes have statutes which establish o an €.lord's duty to rennir those units in which such neint hrs Flrrziy been aenlied and is allowed to noel - creating 2 lead hozrrd. L few juris- 31. dictions nrovide thet in sore circumsterces, u tenant may repair certain deficits in his dwellinf and deduct the cost incurred from his rental neyment. Statutes in hossachusetts and N w York hermit rent abatement, susoendinp the tenant's obligation to pay rent until reoeirs are made. Other erovisions hermit rent withholdine — in which a court collects and holds the tenant's . . 54 rent until PCpClTS are made. Finally, in addition to the neintenunce of mainted surfaces, the nature of the oeint itself has also been subject to legisla— t—t. tien. n 1955, the Americen standards hssociation deveIOped a stELdFrC (thndord Z 66.1) snecifyine thet reints for toys, fur- niture and dwellin: unit interiors should not contain htrmful quantities of lead, limitins the lead content to less than 1 oer cent in the final dried solids of fresh pFint. This ex- cluded lead niements but did not necessarily eliminate other lead additives in the total neint formulation. Additionally, this standard Fs well as the Federal hazardous substances (La— beling) Act of 1960, reouired the saint industry to comely with 55 certain lebelinr erovisions on cans of menufectured stint. RFODE LS a. Because the coste in olved in eeeline with the nroblem of lead noisonine ere substantial, the use of models mey orove to be an effective end efficient eneroach. One set oi two huthe~ u..- metical moflelc eeviSed in 1972, by staff 3. the rational Bureau Fs on aid to determine the heenitude "‘J of Standards we 9 C) zzse and extent of leed eoisoninr in the U.$. in order to ascertain wha£ level of commitment of resources would be rest ennroorinte to alleviate the erobler, one where those resources could be 32. l-J nost effectively an_ied. The nodel relFtes the mfienitude of lead noisoninr in en ereF to characteristics of the neonle and housine in the area. The model is based on the assuention that thos' areas now renortinn lead noisonine cen be characterized well enoueh to nredict Which other areas are similar and thus, (on the averaee) should have the same levels of noieonine. The Primary purpose of the model was to estimate the nctionwide magnitude and extent of lead noisonine, by estiuntine the number of children with eleveted blood lead levels (EBJ: AOMh/IOO ml or more) senerately for Zileetronoliten areas of the country. ft The initial moiel was not CFnable of nredict-ne ESLs or sena- rate neirhvborhoods within a city. The nrocedure followed in the model to obtain estimates of the nationwide meenitude and extent of lead ooisonine con- of 4 tees common to most modeline efforts: '1) F" U) cl" ’1') :1: m 1. data aceuisition 2. model construction 3. model validation . annlication of the model The output of the model consisted of several thles of statisti- cal information inelurinr: * the number of children 6 years of :"r’e and under, the estimated number of hirh risk chileren_(those livins in dil'nidated or deteriorated neusinn) and the num— ber of Edie estimated by the model Of the 241 SfiSAs listed, there were 17 million children 6 years L-pJ of use and under, an estimated 2,000,000 0 thich were Lien risk, with 600,000 EBLs to be ex ected in these SKSAs. * a mini-tire oi" the {35 .f)E'~ESAr: havine t? e. ,“l‘é‘f test nre- dicted incidence of Lt}, in or or to illustrate any ooesible seoyranhic t ndencies. (fire Firure 4 and Wahle 4) 33- Firure 4 soninc — lredicted 25 JLSfls . l. ilt‘l cf flitiODWifie Inciderce of I Greatest Predicted e? 6 based Paint Poi- Incidercc ‘ ‘S-J‘. l l 0 0 '2'“- 1 ~“' A ‘ " I , ' I 1 \ u " I ‘ r l u it own a. n . '. i I ~ ‘ I | .\’ . I O to '. -.’ » : a I \- \t-lt I I I v ‘ o .3 ‘. I s u ‘ E" .J . I . V. . . s . . ~ * .‘1 1 4 1 d C.- r... ‘n ‘ 4 ' V f. ‘ c- t“. ' ‘ I ~ . s‘ ' .. 'P '. . N r E ' " ' ‘v‘ —‘ l "* ' . i ‘ ’9'": J o 7" ' p u -b-4'l. . '-' . c ' - a - ’ "' hi I . ..... ~“ ' ' 1 . r‘ -‘1 ’ ‘ ' I i ‘ ' . g I.V\. - ‘ I (C ‘ ' ' 1'). I ‘. b. - .6‘ . . "2.": ( I. I ‘ ’4. \ ,a h r. o.".; o r -, -. ' ‘ ’. D's.- | - I w '0 r}. ' . V‘ I EL. 0 'c-a-n {‘1 ‘ .>J l ' “ . ~ ~ - . U r n J... . x. I . - t u C no-.- r ' U ‘ ‘ ' ‘ I! '3 I v '1 t ‘ J u ) 0 ~ ,, r'l"'y r'--—1 - I‘ 1 , l . 1 ‘. ~| ‘ ' ' .I ‘ {\— j, ‘n I . “1.1 Ir../ ~ " L- _. ' ‘ \ .. . ‘ ‘ A S F l I I- U A ‘ I J I ‘ I v ' I ) nu. I t‘c .- f“‘ .._ \ ‘ o‘.‘ 4 , ~- L.» . ‘ I' . ’l ‘ ‘ . J I ' f I f ‘ I l.- l‘. i 3 ‘ .' ' L“ nu-“ J - I t I I r p ' ulv'uxv \I ' I | A S a U 3 I f “ 'I.'-V ' A 7 ,. |.\IIIH\|\I|‘~ -. ' , ' e (g . u' I. ' l! . ‘ " —-- - ~—_ \ 11' e. . . .... '~ - t- R I ‘5'-.. f .. ‘\ s ' \ . ' a 13-: \M’,’ . \ l I -, v | I _ ‘ I t .. ../ n l -l ... t ‘ I . . : h 4, - ‘~ . -. 1, ’ '.: \ .’ ' I " \Ql ' .’ - ,1- n} n .1 . ... ‘ ’ I __ \ '1 Source: Gilsinn, Vifieewe "yr-"‘1 -—.—..—.. —. fa fl YOYAL' / -' lrnrmcvm mun-tn er voums mm urn www.m- N‘OU'JNCI U’MTS ' 9 I i . l LN L--.) i 1 l 1 ! RANGE 0")191 [ficmefiéfivkm't 0 000 (Human (3 1.000 (HILURM ,... / \\ / , I I 39.000 6.6.10! m —- ~~.--..-- _ m*~——_—_ --“---v ~—— - t wsouw mum nun 1m rmmnm l DlAPlDAI’fD) .3233 f] 23'- 000 must: U OOJKN) I'wxs \-_..-- -_ _._7- 1.0F0.000 HOUSIS no. 3 ll2, 17 Q _ r' -‘:':- “ ‘50 <‘n "Q- ‘.A J. '7 ,- A, J‘ 1'.- 221310 4. 29 ti hm: dltfl t11~xjcectest Limieer Oi fails SHSA 1U”. PopulaLion Rank 1. New Yerk, N.Y. 40600 1 2. Chicago, Ill. 24100 3 3. Los Angeles - Long Beach, Cal. 14600 2 4. Thilxulelrdiia, It}. 144CH) 4 5. Detroit, Mich. 14200 5 71. 53:. Ixiuixs, DhJ.-11 1. 1 lSCfll 10 7. Pittsburqh, IJ. 10500 9 5h Ike‘nisu. Tom. 9700 13 9. Boston, Muss. 9400 8 .10. Dallas, Tex. 52600 16 ll. Atlanta. Us. 8100 ' 10 12. Rnltimoxc, Md. 7800 11 13. New Orleans, La. * 7200 30 14. Sarl Fra:u:isscr«lnklguri, (kil. 70CK) 6 15. Newark, N.J. 6800 14 16. Minneapolis—St. Paul, Minn. 6700 15 17 . Cincinnati, Ohio-Ky. --lnd . 6400 20 18. Cleveland, Ohio 6400 12 19. Keishingtcni, D.C.-+k1.-Vfi- 6300 7 20. San Antonio, Tex. 6000 37 21. Kansas City, Kan.—Mo. 5800 25 22. Birmingham. Ala. 5500 43 23. Seattlemfiverett, Nash. 5300 18 24. Indianapolis, Ind. Sl00 28 25. Buffalo, N.Y. 5200 23 Total 253400 ~-—~o—---. ---_wrouu~p-m—.——--. ‘Ww—_” " Source: Cilsinn, n. 114 * sit tee of? the number of children in each 1:31.311, .ith blooci lead levels of 404m or more, 50 or more, 60 or sore, end 70 or sore The estimates yielded bv the N33 model, confirmed that nedie— tric leud noisonine is a Ina.jor uro n heelifl rrobltm in this country, as they sueeested that some $00,000 children if tested, H . - . . 6 . . . mould show undue sosorntion of lead? The authors of the study ceution: ...it is believed that the nrebdem will nersist es lone es neeline lead sainted surfeees are ec— cessiele to youne children. Discoverine and treatine children with BBLs will nsrtially alle— vivte the nroblem, but its full solution re uires the renovel of leed neint on all suri ces accessi— ble to children.57 A secrnd model, devised in 1372 hr Juenite Heston (KSU r of Arts, Densrtment of Geoeranhjj tnk s a different sch. It is a snatic l r? ther than mathematical model, and it is to be used on e comr: unity-wide rather then nation- wide scale. The nurnose of the model is to ascertain which children are at erratest risk to lead noiseninn by defining and then mannine by census tracts the areas of a city (in th icnsinn mes used) with low incomes end old deteriorated nre-Jorld Her II h-u_s ins. ”he model was intended to address the oadvsntupes nosed by the three most commonly used risk-deter- minetion annronches: l. The reneom selection of ectients from nediatric hos— nital wards or nnticnts from neirhborhood health clinics — Tee enjor disedventvee'of this aenronch is that the most denrived and disorrcnized femilies do not use health cen- ters, thus ccusine the child livine in n hish risk area who does not come to the health center to be the most likely af ected. ”sis of nfint from the child's environment - edvzntcse of th's Cnnroach are first, a d rovine from a house conteirine lead ncint so of such print mould not he screened, end chine used to do such screenire is suite 34. ezr'ertrtve €116 iflNJS Ln1e07¢unric2fl for~z”oexs scrv enicdf. 3. Tciehborhood door—to—door leed noisoniro educationcl carneiens in vhich femilies €1wv1xr~ed to hrjre their chil— dren to heclth centers for lead reironi1~ tests ~ The dis-~ cdventeee of this Unnrooch is that sore 1r"enilJ'es m3? HOt hcve trensnwrtntion, or cannot efford to take time off from tork to hove their children tested. Gretcn's rodel is hesicclly constructed by ninneintins in Shoes the residents ct risk. (See Tieure 5) “he stens involved include: 1. coretr*ctire tehles 8rd tens of t”e "orcentcee of residential structures built before 1940 find the redian income by census tract, for the uholo city 2. connifiction of urns in terns oi fire of the residential structure end tre condition of the struct“re for all struc- tures in each of the census trects screened intensively in the hieh risk arecs 3. the use of certain veriehles celled nredictors to in- crecse the nredictive value of the model including: e. n historv of mica b. existence of anemia c. number of siblings livinh ct here d. zwfiritel snxrhis e. sunervision of c f. source of familv incone E?- 1’1‘10unt of tine child is left in the are of others The edv0nt? es of the CnPtlEl model es nronesed by naston are: it includes ell children within a nre—sclected Cree, not just those visitinfi clinics; the chilfren MP“ be tested in the home by a finder stick blood test, scvinr e femily t‘cnsnor- tction costs and velueble work tine; the “nrrOCCh has only a minimal cost, szinr it econoxiccl for-massive screenins.58 Althounh detection is only ”helf the b ttle” Crcinst lead poisonine, rodels srch es ttesc can Cid states, cities end inn ene conductine intensive local ero— commurities in develon Prams to acconnlish this. “IQ. - -' “-0 *.--~u~o.ooo.--w CENSUS TRACTS RANKED ACCORDlNG TO RISK ! l’-_\_/ 33.07 35 $ 1 I. u- ‘3’. f, I V _ . I, , I ‘ 1\\‘\\ |' '\ '\ ‘ \ '..“‘\“) ‘\\\“\\‘\‘ l 3“ \ \ A. , ;' " “"3" . Q; u. . =,¢M H , , f H I\(‘\u ‘) ‘_. v.0 I i ... , , j" ,, ,, .AL \__......_.._._ _. ‘/. -— ,.... r...“ 2". r '\._...-_.- 36 07 37 K79 ') 16 0| HIGH RISK AREAS - 2". _ 0 such 1960 C€npus auflc .,.. _-. —.... -———--. am-“ _.".‘R: I m1- 5—— Fifiure 5. Census Era ts fivnkefi nccorfilnr to {18k gource: fiqston, n. 60 (338% ATUU‘LS Es entially, a pivcn city's nrorr1m for fierlins with lead hazercs consists of Mh'ee carts: identificetion, includinfl casefindinfi; environmental control, incluflins treCtment of ‘victirs of lead intoxicati1n ; and elie'rCtion of sources. The *‘S (‘3 e re six necessC.ry colicies to be incluecd when ini— tiatine C Cd— control hroarLu. First, the chief ,xecuuive of the city Covernmert rust actively suocort the concent, as the croeression of activities will inevitably lead to involvement of a nurbcr of int~e—city ove. W1ental (enortmcvti. decond, it it must be underrtood that success ul 10-6 control meCsures will intiCllf increase in cost as more le 6 “1""*'c Cre identi- fied. Sri r6 there is ittle VC l-ue in C‘hTOhri.t “n funds for casefindin‘ of one 838 eroun for one year, Cnd eliminating the funds in the next veer. The fourth nolicv is that ececific Foals must be established ct tne outset, co the t tee evaluation nrocess used will :ive a true nicture of the “rocrer in erosress. Short rCnre coals would include: or'”11 cticn end st u.fiine of the evency, linkinfi neiehborhcod orecnirations end other com- munity fienarteents, berinninfi co munitv efuCCtion Cnd screenine in effortsgNthe svsterCtic selection of FCO*PP“th Grove to he tes- ted. Lon: rCnre so: s xivht be: lsrislction Cnd firm ficleecine and renovation hro: ems. The cstobl1shmcnt of clear—cut objec— tives Crc review of sricrities is 'he fifth er entiei_ n licy, as if such objectives Cre lifiited CD6 re: listic, the nrotecilitv of continuint fiscal succort is ercutcr. PinCllv, each city must develon Cnfi follow C mien of Cttfe ’ Gcsicred s“ecifiCCllv for its individual needs and resrurces. A snecific sequence of stews Cr: recesrCrv in “lCnninc and eicecutinp a lecd cor l. EWM3 "wrinm .m Cm elite, must hecozn f st convince th .f‘ ts of deelinr m tionally, thr acces to the (EiW', ("'1 2. The estrblis size of which een set for the eroer finnlized‘within 3. Mobilize of lead. f. Cesefi attcck 5. effort. {17.2”} «iimw .1 ( u-A-‘ , the lrrrer Wr‘ .1— CthiniNE 50: The eradication Jhile slum cleertwce for the moor is the DOiSODiF” cup be ruc detectitni, trettrmrfl; moval of lose fr have demonstmetefl In Chicveo, the poisoninr onercted t Ciels, he l_th “ON er in 1955 and by 176? over 1500 treated. GJCVQted bloee lead rer cent in 1007 to of Ar‘ \/L S in the member Ifi?te. Ir1 lo2 203 cases cf 700d cit, \.; r {101.1530 {-7 +11. trol “roerem: overs” in tie city, inclueine ehetto resi— e concerned about the wrehlem {37 must 9 10931 r"“"\f£‘.1”‘r.'£91’1t thet tee econonic bene- jth the “roolem, “9?“’nt the costs. Adfii- ector cf t}1e leJc—control rrorran must have chief e} meCI uiVC. 17.0 0 hment of C staff for the less nrorram, the ends on the size of the city, Chfl the reels ’T’l. A time —ohased ,ct'cl ol-‘n Should be three months this steff educational effort concern‘ the hroblem . .~ 1. S . but tool to tie environment. total reind 13x t it lecfl in r in nu” u.- keenin hroblem disintive enfl fiecsl sueeort for a of eafi neironinr is not “n i: oessible t sk. I" with erovision o: edeeuFte hou“1nr combincfl most effective means of elimihétion, lea c1119 cess . lly reduced throueh efiucatiOh, early emé follow~un ororrtus 0 include re~ ‘ vherever it is founfi. A few cities vzlne of such methrés. re is C mas;ive screehihe crorruh for Feefi Wroubh the co Joi Fted ef“ort of local offi- 0, find the conmunity. Uhc nro~row was be.un over 190,090 chilére* hid hcen testefl no lkxuuise Of this DTTVWTU , the incidence levels {‘non” eri lriren _..\¢ 3.fl cent in 19b3. Alone with the rise 7701‘ 93 99 99b39y Cffie U decline in the fatality first F93? the 9180:”e WES Knee rewortablc, 'fiiS nirwti ere ifiviortnf Kitn fr~m:1it¥ r) A (N rfite of L.U her cent. In 19c , the number of reverteé creec rose to 707, while tre frtality rate cronneé to 1.? rev rent. The Chiceeo nroere: iv concentrated in nine (rein in the city enfi inclxmflnz'the onereifiCN1 of an.rmflmilutory tiwwrbnent cnnwter.60 pimilLr ficuree hwve been ren rted iron New Yorl City, tnrounh e nronrcn in which physicians from all mc.ical ceenciec in the city are encourvrec to eenc blood encci one on all sus- nected arses of lead noisoninr to the cit“ health cencrtnent laboratory for n onnt vnc accurate blond lead anilysis. In the "leafi belt” “rcan of the city, nublic health senitcriene visit ever? houee in Cesirnetefl bl eke. (flee Ficure 6) They tLLe scraeines for 7eC€ écterminntion , in uire ebout nice, tcke uriLe scmnles from all children under 6 years of Lee, and ac- vise ncrents about the cenrers of nice and its association with lead noieonine. irem Jenner? to July 1970, bfi,54& blood sneci— mene were Cnelycefl by the New York Cit" 'eFFth Vehirtnent, ”0‘0 children with blood lead concentrationn of baflr or erefiter were found, enfl 2 lecd noieonine ccueec deaths were rewrorteé.61 x'fe U renortable dis— J C) \ In Ihilfidelnhit, lead noisonivw ma ease in 1053, hu‘ with little effect. In 1 en, the Philacelnhia City Council took action end reeulatinne were ieeuefi rerarfiine the labelinr, ennlicnticn, and renoval of 19:6 neint. In lécQ, a urine screenine nrocram for lead noisonfin’ rwcn" chilflren l 1 to F years 016 was initittcr in Philudclnhia'e hieh risk areas. (See Figure 7) Thronflh tiese efforts, numerous nronerties have been mcie Ser, renorted Sees of lead neieonirr hfve increased, Elrul the-'wurluyr of :LCCG.1T31FWUliHF rieetiy” herirlcCIWN1305, Baltimore $33 one of the first cities to recernize leafi TDOiraoninF in chileren Us a eublic heclth nroblen. For over 38. Repofled Co ses Of £3119 POISONWG - ..--__—....,___.—_ .— 8)’ Place Of Residence ; New York cny -—w¢3 .... ,.- . _.v . a" LEGEND 't'a-n a High 'ncédcncc Ava-'35 Q lwio'ed Cases AfTER JACOBZINFR AND QAYBIM, 19¢; ' " "" -‘-—-—4-—— .— -A.——-— a-a——-,._—.—— A-.- .— Firnre 6. City of New York — ienorted Cases of Lead Poi— eonine Source: Gaston, n. 95 ,--.--—-- ~ ~-....—.-ns mmmwnm;-~~-m. A—A -—- —- 1952' a“ ZNGAll S. \ AFTER \ \\ LJ ii” C .q: CL LL .J Spot map of 219 cases of lead poisoning by Philadelphia residences of patients; 1356 -1960 y- § t i 1 4 I 1 l i Fieure 7. Soot Koo of ?19 0930? e? Vere Toironinr' by Philfflel hi? Reeifiencee of Patients; l9fif-1960 Source: Heston, n. 2? 3O Yeflrsy the city has dealt win detection and orevention nroereus, t-eu health and educational connCiFns. Association, the city tion from landlords. nrosrers alone with n: .4 6 '1' IV *n'. yielded encouror ent nroeress is that environment. hsvardous sources have been reroved, helned to find Cincinnati has attemnted to deal with the coynlpx case-findine, by the establis‘ment of US troject of vario End a in mrnv fields, The Center mointeins Poison Inforre 1" OJ diarnosed for insurinr adcouote vidine tennor:rv \ f“. r- C, (mi) records of known V a sienificant role in In 1909, the Urhen Le; ." T 3. 111') O f :3? Q ; 1 t j to the I (1 qr: . (NJ- U~L11\) U housine codes, re no child After hosnitolization, lead-free nrevcntion, cit? onencies, a tion Center, lead eiurtodicl_ CS fo‘unterv citisen action :r Gomrittce r‘11 the erohlem throush continuous J. include enforcewent of eniderioloeicel surveys, erd intensive 'aith the hole of the Irooerty Owner‘s heelti desertment hos received coonera- The continued existence of the various int removcl from Sony dvellines, has silligs. W Another k~v to Unltimore's annar— '3 ever returned to s denserous the child goes to a con- returned to his f“milv until all I1 or the ifirily hes bee housine. ities of nublic and orofess'onnl education, '7) t... Control Center, a co.eorotive institutions, erofeseioncls variety of other interested individucls. close relfitionehin rite Gincinnati's and is rcseonsihle for renortine all noisonine to the teelth denertnent, insnection of victim's hetes, for tro- CCfre, and for mejntf jninr‘ ’t’TT'OfiCI‘ - . . . , 67 one infurins C eoufite lOllOW—UD care. J in several cities hes also slaved G r‘. I... the iisht to eradicate le ooisonine. cue of Rochester, Ken Vrrk, nereuedcd e s ?rofiect Uniiit Youth Incentives Prcrrum fei'Lfljientiifi(é Infl‘mfmtixrl in its study of lead noisoning, ti ouvh the collection of stint- ssmnles from slim herbs. the erorrem has continued and the Youths new work with the Rochester Keiehborhood fleclth Genter aid with doctors from the University of flochester's Strong tenorial Hoseitel Deportrent of redistrics. In edditien to col— lectine ruith semrles, ifiu31youne neorfltanyiosa 3||~5 “\IW‘CYlinjj ixu li“'fi n(\t fu'xiu I it} l 1‘1 , n.,. in t08p13d13 awd .Ylnlca .HJII merceninq effort weal] isrtcning cffdlt H‘ifl AUTch, Fvull SCXCPning. in L(.pitnl$ "mall screening effort. Hues screening, 12% of all thildren screenwd SCTCkhiHS in 9e10:101 arcna i”§?31 so: Ve}', Serxll 3(l ceiling; of :oxt Iv ling in howpltnlu l‘ilot lirojcct, hxitintrul hv ctwtavnity' groove In ’H\5pitill , 3~lnvnninq tilture 1 {tort:; In Tlinixi Ferorning in Nodel Clries 5! TC” 5' ,.___ -..———.— » --~~-- ”.-.-.....- .. -—- —----—— .w‘“ -._— —_- ,- ~...._ ....~.. . .. :;rcering ---__ .. ...____..1 A-..‘ I C4 ! | l Primarilyo ——.-— “ham—-....— Continuefi ‘ . 1 Area i fi::f:::l Yrur Descrigticr of PY05er ‘ L _, - --__ __-._._..__-_____._ ______-.- _. __...--- fxiluaukcc { {vat thldlon with Lead Poisoning Early / Treatment Lead Exposure Firure 9. Crnccn “n6 Comte“ zoning 91* 1oteniirl Sites Source: flurfié Deprived Children Transfer of Small Children From 4—.— Prevention ol Pica Pica \ Case Finding innicertifil 'piii12lzoofi for lntrrver ti {NC-3 (crimes, T). 739i (1 Poi— 1 . - ~ - I q ~ II'A- —\ yea J- 1 ~'( . - .~\ ~ I ‘ - ~. '..I ~\“ .3 I ‘ 1n phone ChliFFGfl nnw _uc. 2c :1? 1% a Pfrco c s "UuHClD'C. " ‘ ‘I' H‘ '4 rm -: I . . '-~‘ ' \ 3 \”.J‘ ~ ' . . v\' . .lr 1. .I‘ V . v lrlmerv res? H;;OlllL¢ far as urOl oi nlca 1103 .Ltu fire fgm113, 0" ‘ Jo ‘ . . - - J- ‘ . in -' . V.“ . ' -.- 3.. q I-‘ a» - I ~ . ra o N 3nd U'U? izrentrinunzs cc lflfOTmG? oi ch etn.crc economrtcc with this hibit. nose refiin, school narcnt FTOPUS, :16 commu— rs nity mcctinfls shoulc C11 be utilized in iris CGUC‘tionF1 effort. Burce and defines Cleo reconmcné for sne11er ccrwutities, the establiecnent of "lead centers” - sccciCI clinics Vierc rich risk children receive evaluation can care. “hose centers should Ibo comnrisod of an intordiscinlincry staff to_he?n femilies cone with environncntal situations which nroduce tn: rrrceurc hrzurds .. .g n‘ ‘ 1" o a. . A. . -,;J- ’ o . o‘c‘ "(fly— - ~' - . a one n30v1cc co:rnrfig,.u efiucntlcn of L (‘11.flfii: tnrnnrh reieonal (‘3 contact. The efficrcv of ruch centers nil? ultirctcTy dewend . - . .. . . . - b7 unon the locatlon End 0339 filth whlcn hFTOPtC can Othln HOLD. “ nccorfiinf to tnrllon, a two—fold tssk exists for eny com- J munit“ th~t in consjcer‘nn the estrbliekrent of In Cctivc leer noixon'n: Control nror~cn. First, the chiifircn ct frertest risk nficctec. Second— ‘4- must be locatcé, screened, find trC'ted crew J, (n0 ecrbfns Kore imnortfntlv, vs fer US the overcll eliminfi- tion of lead h0i80hifl“ is concerned, the 70‘rce cf the lend— bccefi sci t must be iocafed ape made uneveilcble for further incestion. ”It 17 obvious thtt any wvcchn desirnrd to control or elimin to load—based ntint noisenirfl in e cormutity nuct be bruscrf or1 {' lilo Henri; Iznrleiés‘fizrufiiz" rof' true rviif‘efirioirfievelgwwzhotho6s CH” ifiontifjdru* roor~1wmxsirr and lead—inforted units. 101 Ckianncls znfl 00‘ unit* ore. SUre must combine to dcvelon zcthods of removal, cnfi the forcinr of 10ndlorés to Unh016 00608 81 ovfito1v desinne6 to include testing of “c.1nt 1£y01s. {elooztion services crn servo Us 0 bcsic tool bnfi only if oomhiao6 with RoodUite xetho6s of sorennin~ anfl ce- curin? the new unit. bnifir hovsinv an6 invest rnfi ovmctice FD nust be abol shod, :26 the behcvior r nj.fesL 06 in rovernmontal and othor struct“res that nernetuau es such condifii ns must be .72 nc11y, the rolo of :11 levels of FOVG“”K€Tt is L130 key to Uny efforts m11ioh seok to erafiiocto 1036 roiooninr. In 1974, a firoun of 200 rovernmrut oificit1s Lnfi Civic loaéows met in .Jashi-n ton D.U. to 6ruw unfrOUOSD Is for nrovcntive measures. The aroun's rccomm ndations were: 1. Lsto )‘z1iszthin'T 0.10301‘ '~.'-.-'02“1.£i31.* 1‘6.faitionsz‘vwinr; ‘oe't‘rxeen Cd ral ro»ornxcnt unoncies cné or Vtte hosnitalg 1‘") 2. 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(......m...) ............. :. 1:...5 . o._-_..~.f..a -M.~.—._ .v"—~ _ *4 . u . . . . :L..!.:;1 1:;>;;.:: L.?:: ”0.2...." «... ......L/LLL. ...—w _....h..__. ..m {IT/“1...?” ...»..ZJIL 1...”; M. II.;/.1.:..; 1.L: Ix...:.unx.1 n . . . LTZTLLZT... .T. :2. U...— r..:..... TI... ~.........m..”......._. A” I“ va.fis.z..u7. .. . ..t:+;: t:::...::.n:.; :r:; /. .2. ~ .—. . . Aw: .../...m_vnw....i-__ . m..~.r..r A ~o IT'V“ 1 is. L , 1 Fay-w-w- g . i . «p.174 1791'.“ " . .‘ .r-: I. I.“ 'v'mTV‘u O i 11‘: FY" ' 4 4.9: 1:25. 7.5,: 2;: .2; 132:: 3;: a; .32: 7.‘..:m_;;.2;: E: :3 ......L 3:" C .3 3:5». . 0 -a1 ”1.. 1._.__..::.. 3;. 4t: .25». 1:7: .3 O c “ it .3.“ 7.2.3; .. ‘ ...c." ..C:€..?.i.? .... .42.; 1.4.2.95??? S: 2...” :42. :25. 75.: 3: .3... ~....\.::;:__: i:::.:... .92“ E: ”1.5- _3:....:.r..5 .5... 5 13.3.2; .7 :33 «3......73 :3: .z.:.._:..:._ 2:23.” . o r u p . u a :27 “32...; at... ’53:: ...1.m._;._.va L35: — ... P I u . a: W.ID...a-/ .2: ....9..:7 9:; .. h D .. “....- .urrf/ “3.7..“ .ZT Lt.“ ....X..::...~H~TH sf: .... 5.3.4 L2.“ «LL? : 1.5.5.. 3 .2... ...L... in: T S. 2:7. .3 ,::7m_,....£ ...: E: C2“... 3 7.241.?253 ,3 E _,....H\.m.:..;:._x {3.5; L $22 .3 7.35.». 15mm $5595.23 95 2: 3 22:3 .2; ~12” 2.2: .23.». 15.1.: 9.: .2; 32:.C3mqfifi 139;... .L «o: 8).. v.3” b: H at: ..C .1.__..T_.f..:.~ .z: 2:. his; .5 75:5; LEE: .3 S ~...5..:.::_:~ .393: 2 P55. ... do...“ dun HM C“) FOOTfiOTbS 1. fihite, on. 1-2 2. Ibid., D. 2 3. Hoar (in Coulston anfl Forte), n. 76 4. Lin—Fu, n. 1 Committce on Environmental E 0‘. U1 o Greer, n. 248 7. Subcomcittee on Accidental Poisoninfi, n. 2?? 8. Chisolm, n. 21 9. Subcomxittev on ficcieentfl Poisoninfi, n. 232 10. Ibid. 11. Ibid. 12. Committee on Uiolorio Effects of Atmosnherie Pollutants, n. 135 13. Lin-Pu, no. 5-6 14. Committee on Bioloric Effects of Atmoeoheric Pollutants, n. 135 15. Ibid., no. 139-140 lb. Lin-Eu, n. 12—13 17. Ibid., on. 14—15 8. Lin—Eu (in Chiifiveg Jon—Feb 1970), n. 5 19. Graham and Grnhbm, n. 39 20. Ibid. 21. Ibid., n. 38 22. Ecrncs, on. o—9 tv-l 23. Committee on Hioloyie Effects of stmosnhcrjc Foilutauts, on. 206—207 24. Ibid., no. 200, 20* 25. Ibid., n. 209 26. Ibid., n. 210 Hear end Aroncw (in Uoulston and Korte), an. e L_J -97—900 ‘cllutfints, c. 139 K) «3 O E“) on 9—4 3. Committee on Bioloric Eff etc of Atmocnheric \Q Heedlemen et 81, no. 245-243 (MR) 0 COLLittee on fiioloyie Lfiects of Atmosoherjc Toilutunts, n. 139 1bié., n. 140 (J) 1" I‘.b~ '..J dron arfl Stofen, no. 11V—120 {\J H O Tenner anfi Kev n (13 Couleton an? kortr), no. 152, 154, 165, 189 W W be) 0 34. «alfron 6nd Stofen, n. 191 . 1bi6., n. 175 16. fiilfljann et 81, Two. 330-231. 37. Fulwiler cnc Jrirht, n. 374 38. Ibid. 39. Bernbko, on. 1737—1739 40. Browfier et Cl, n. 47 41. Ibid., n. 48 42. Ibid. A3. Allen, n. 90 £4. Browder, n. 207, 200, 211 45. 3nbcom~ittee on AccidrntLl Poisonine, n. 207 46. Comiittee on BiolOFic Eiiects of Atmosnherio Pollutants, no. 2o4-Qo7 ' 47. Ibid., no. 257-258 48. Ibi6., n. ?63 49. Holman, pp. 592-593 50. Greer, no. 260-263 51. 33., nn. 4, 6 52. Greer, fin, pAo_955 53. Ibid., n. 755 54. Ibid., nn. 556-259 '5. Committee on Biolonic hffects of Atmocntoric Pollutrnts, on. 75, 78 56. Gilsinn, on. 13-20, 102—10 570 1016., he 1“,]? 58° Gfiston, ... 53—61, 91—v2 \J'] 59. Subcomrittee on Hetlth, no. 76—78 60. Cormittee on Qiolorie iffeetc of atmosrhefiic Pollutants, on. 135—137 01. Ibid. . Lin—Pu (in Chilereg J n—Feb 1970), n. 7 64. Lin-Eu (in Chilcren Jan—Feb 1970), on. 7-“ .'\J 65. Thomas et 21, n. 103 06. GChCIT, 7N1. 30-721 67. Burdg {nfi 70:10:, h. 7K2 63. Challon at E1, o. 659 9. (Hufllon, 7T7. 561r4h32 b 70. Chisolm "n5 Fenlvn, n.<£“ 71. 72. 73. 74. 75. Lin-F11 noore, (iralyim Lin-Fa fiuinn, Ibid., Graherl K1 (in mrujlr‘Lgfl Jan—Feb 1970). m. “~31 UH. 1430—1434 {qy- CIT U“m, T7. 41. (in Uhilflren Jen—Fob 1970), p. 9 Duncan, 1nd Cox, no. 4—5 n. b 1.116 Crnhrxm, n. 41 1213.7:(1‘ (1 uit’IIlf ilffILFJEj Allen, John 3. ”L636 Lois oninp: Lew Look 3t up Old Hazard," To— EGE'S Health, JCfluC_y 1962, UN. 22+. Americ n Ace demv of Peéietrics, Cor121ttee 01 :nvironmentol F37”r6s. "Leute and ’hronie Ch116h006 Leah Poisoninfl." PC6L tries 47 ( "”511! 197 J- ). 9 (4’ I) O o American Aea6emy of To e6iatrics, Subcormittee on Aeei6ental Poi- soninr. ”Crovefi+1 on, Diaenos 3, 0R6 Trea.tnent of Lead Poi— sonin" in Ch 116h006. " PeoJLtriee Al (Aueust 1C’“): 291—298. Barnako, Donne. "Chil6hoo6 10:6 Poinonine." Journr1 of the Ameri— can Le6io€1-.is.o .O .’ ‘ v0 O .'_O |~-. .o- n~---». .. . I -. CI . ~ I I -. ." .- ~' ‘.--. .' --.o... I o I -'-. .~‘- I ‘- - . '_ ‘0 UV I.‘.l I ‘ u-'- - .. » § 0. - . a - O I -' '1 I - ‘0' u n v. I u- .I- -, t . , I - I I -. .-. ....‘.‘..‘.'."'..‘ . _. . ' ' ' .' ‘ . ' ".‘ .‘ I .__J-.~>-I--. 0- ~.»_. . I .' .. ... i .- II - ._ .. . - .. 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